David:  I'm still using the same Medpro Prostasol capsules which I bought about 3 years ago.  The only new products that I'm taking are Vitamin D and Triveratrol which may be causing the decrease in PSA.  Don

From: "tinycamera1" <DavidCohen99@...>
Reply-To: natural_prostate_treatments@yahoogroups.com
Date: Wed, 08 Jul 2009 02:30:15 -0000
To: natural_prostate_treatments@yahoogroups.com
Subject: Re: [NTPC  Yahoo Group] resveratrol produces longevity and can kill cancer (??) - (longevinex)

Donald:

I had the same experience.  The new QP+ seems to be 3 times more potent.  I only take one every couple weeks.

David

--- In natural_prostate_treatments@yahoogroups.com <mailto:natural_prostate_treatments%40yahoogroups.com> , Donald Lytton <dhlytton@...> wrote:
>
> Hi Shel.  Your e-mail reminded me of something.
>
> I posted this e mail 12/9/08.
>
> "Hi :  I had a prostatectomy in 1991 and they didn't get all the cancer out.
> I started on PC Spes when it came out and now I'm on Prostasol from Medpro.
> My strategy is to let my PSA drift slightly up to keep my testostrone at a
> reasonable amount ( betwwen 500 and 600).
>
> Last spring my PSA was 1.1 on 5 to 6 Prostasol capsules per week.  I stayed
> on this amount and expected my PSA to drift up to 1.3 to 1.5.  Much to my
> surprise, my Psa, taken on Nov. 26, 2008 was undetectable.  I went back to
> the lab to check my testosterone and Vitamin D.  My testosterone was only 38
> and my Vitamin D was 73.9 ng/ml. I take 2000IUs of Vit. D per day.
>
> It didn't make any sense that my testosterone should be so low on the same
> dosage of Prostasol.  It's possible that since I'm 78 I'm producing less
> testerone.
>
> I'm only going to take 2 prostasol capsules per week and see what happens in
> 3 months.
>
> If anyone has an explanation, I'd love to hear it.  Don Lytton"
>
> Since then, I've dropped it to two per week and then one per week and it is
> still undetectable.  I'm now taking one capsule evry other week and I'll
> report the results in 2 months.
>
> I thought it might be the Vitamin D, as I had been taking Longevinex for
> about a year earlier with no unusual results.  But about the middle of
> September 2008 ( 2 months before my Nov 26 PSA test), I started taking a
> supplement called Triveratrol Plus sold by Dr. Julian Whitaker.
>
> Each capsule contains 150mg Aloe Vera extract, 100mg tumeric extract, 50mg
> trans resveratrol extract from Japanese knotwood root and 40 mg red wine
> polyphenols from grapes.  I take two per day.
>
> I have no idea whether it's the Triveratrol Plus that's causing my
> undetectable PSA, or maybe it's combination with Vit D.  Or it could be a
> fluke.  Unfortunately, you can't prove anything with one person's data.  I
> thought I'd throw this information into the mix.  Don
>
>
>
>
>
> From: shel litt <shellitt@...>
> Reply-To: natural_prostate_treatments@yahoogroups.com <mailto:natural_prostate_treatments%40yahoogroups.com>
> Date: Mon, 6 Jul 2009 06:32:25 -0700 (PDT)
> To: hope <HOPE-list@yahoogroups.com <mailto:HOPE-list%40yahoogroups.com> >
> Cc: PC-ACT@yahoogroups.com <mailto:PC-ACT%40yahoogroups.com>
> Subject: [NTPC  Yahoo Group] resveratrol produces longevity and can kill
> cancer (??) - (longevinex)
>
>
>
>
>
> Has anyone here tried Longevinex?  (i was told by someone long ago that Dr
> Myers recommends it, but cannot recall context.)
>
> -Shel
>
> Subject: Dose determines whether resveratrol
> produces longevity or kills cancer
> All followers of resveratrol science take note! In
> the study abstract below, researchers describe how
> resveratrol affects the cell cycle, that is,
> cellular events leading to its division and
> duplication (replication). The S-phase (synthesis
> phase) of the cell cycle is when DNA synthesis or
> replication occurs.
>
> In modest doses, resveratrol causes the S-phase cell
> cycle to proceed in slow gear, giving more time for
> DNA to be repaired, producing longevity. In mega-
> doses, resveratrol accelerates the S-phase cell
> cycle and increases cellular death, thus inducing
> mutated tumor cells to die (apoptosis).
>
> Advice to consume mega-dose resveratrol (more than
> 300 mg) appears to be ill advised. Other studies
> confirm that mega-dose resveratrol (as little as 360
> milligrams for a 160 lb human) shortened the lives
> of laboratory animals on a standard calorie diet.
>
> The widespread false notion that the equivalent of
> 1000 glasses of red wine is needed to produce the
> same effect as pure resveratrol in the laboratory
> should be dismissed. It has been shown that 3-5
> glasses of red wine, providing 180-300 mg of daily
> polyphenolic molecules (resveratrol, quercetin,
> ferulic acid, malvidin, catechin, kaempferol, gallic
> acid) produces unusual health and longevity in
> French wine-drinking populations. Copyright 2009
> Bill Sardi, Resveratrol Partners LLC
>
> For the full text of this report, email Bill Sardi at
> bsardi@... <http://us.mc343.mail.yahoo.com/mc/compose?to=bsardi@...>
>
>
> British Journal Pharmacology 2009 Jun 25. [Epub
> ahead of print]
>
>
>
> Induction of a reversible, non-cytotoxic S-phase
> delay by resveratrol: implications for a mechanism
> of lifespan prolongation and cancer protection.
> Zhou R, Fukui M, Choi HJ Zhu BT.
> Department of Pharmacology, Toxicology and
> Therapeutics, School of Medicine, University of
> Kansas Medical Center, Kansas City, KS, USA.
>
> Background and purpose: Resveratrol (RES) has been
> shown to prolong lifespan and prevent cancer
> formation. At present, the precise cellular
> mechanisms of RES actions are still not clearly
> understood, and this is the focus of this study.
> Experimental approach: Using human hepatocellular
> carcinoma-derived HepG2 cells as a model, we studied
> RES-induced changes in cell growth, cell cycle
> progression and apoptosis (death). Key results:
>
> RES at lower concentrations induced a strong but
> reversible S-phase delay and mild DNA synthesis
> inhibition, yet without causing apoptotic or
> necrotic cell death.
> At high concentrations, RES induced apoptosis (cell
> death), which is mainly mediated by the
> mitochondrial pathway.
> Overall, RES was a relatively weak apoptotic agent.
> Mechanistically, MEK inhibition was identified as an
> important early signalling event for RES-induced
> apoptosis. In comparison, activation of CDK2 and
> checkpoint kinase 2, and inhibition of
> phosphatidylinositol 3'-kinase/Akt signalling
> pathway contributed to the induction by RES of a
> reversible, non-cytotoxic S-phase delay. Conclusion
> and implications: It is hypothesized that the
> induction of a non-cytotoxic S-phase delay may
> represent a useful mechanistic strategy for lifespan
> prolongation and cancer prevention. When cell cycles
> are selectively slowed down in the S phase, it would
> cumulatively increase the total lifespan of an
> organism if the total numbers of cell divisions of a
> given organism are assumed to remain basically
> constant. Likewise, when cells proceed through the
> cell cycles at a reduced pace during DNA
> replication, it may allow cells more time to repair
> the damaged DNA, and thereby reduce the chances for
> mutagenesis and tumor initiation.
>
>
>
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> (866) 405-4000
>
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