Dear IMVA,

Below find The Hun Hordes of Mercury. This medical essay makes a strong and plausible case for understanding one of the environmental triggers of diabetes Type 1 and also for the impact that mercury is having on the incredibly disturbing rise in diabetes T2, worldwide.

It is a long read. The first few pages recap the current situation and concepts then dives into a new and unexplored area that will eventually turn the diabetic and heart disease communities upside down. The Hun Hordes of Mercury is a perfect title that implicates mercury microgram for microgram…each 3,000 trillion atoms of it…..as a vicious and numerous toxic invaders that sooner for some, and later for others, overwhelms the body’s capacity to defend itself.

Mercury is the invisible enemy and it is coming on secretly, like the lead panzer division of an army of toxic chemicals ready to overwhelm the human race. Mercury’s destructiveness until now has not been recognized in diabetes and heart disease.

Also we have a new webmaster named Donna and she is busy with major changes to the site. I invite you to visit at least the first page to see our new mission statement. Please let us know your perceptions and any recomendations for change.

And after reading this if you feel like you would like to join a group of people dedicated to fighting this enemy in the diabetes and heart disease areas, please contact me.

Mark Sircus Ac., OMD
Director International Medical Veritas Association
http://www.imva.info
http://www.worldpsychology.net

---

The Hun Hordes of Mercury
 

The number of U.S. adults with diagnosed diabetes,
 including women with gestational diabetes (diabetes that
 develops during pregnancy), has increased 61% since 1991.
                                                                                                  CDC[1]

 

     The CDC says that diabetes is disabling, deadly and on the rise. The incidence of diabetes is skyrocketing not only in adults but in the juvenile population as well. Healthcare experts have called the alarming rise in diabetes and its related complications “an epidemic” that threatens to spiral out of control. In 1997 15.7 millions adults in the United States were reported to have diabetes. [2] By the year 2002, this number had already swelled to 18.0 million or 8.7% of all adults.[3] Diabetes and its complications now claim hundred of thousands of lives in the U.S. each year, incurring total expenses of over $130 billion in direct and indirect costs to the healthcare system.[4] Worldwide, the number of people with adult-onset diabetes is predicted to explode in the next 10 years, doubling to an estimated 221 million people.[5] By contrast, only 43,171 people in the United States were diagnosed with AIDS and only 18,017 died.[6]

 

     If we add the number of heart related deaths caused by diabetes to the diabetes statistics we actually find that diabetes is the biggest killer in the United States. This means that the above statistics understate the problem. Thus it would help a lot of people if we could isolate a basic cause for this disease. Though there are several important causes/factors contributing to the dramatic rise in diabetes, the core cause has eluded scientists and doctors - meaning a cure to diabetes has not been found. This medical review sifts the focus away from typical diabetic etiologies and suggests we concentrate on causes that can quickly bring on diabetes through intense chemical exposure or slowly through decades of low level chronic exposure.

 

Diabetes, which had a per capita incidence of 0.0028%
 at the turn of the century, had by 1933, zoomed
1000% to become a disease faced by many doctors.[7]

     “A growing number of children are visiting pediatric cardiologists to treat their high cholesterol, or seeing endocrinologists to keep their diabetes in check. In short, kids are "catching" the diseases that kill most adults,” wrote Krista Ramsey of the Cincinnati Enquirer. "The picture has been on the wall, but we've just refused to see it," said Dr. Larry Fox, medical director of the Northeast Florida Pediatric Diabetes Center in Jacksonville. "We have to realize that the kidney disease and heart disease we used to see in people in their 50s and 60s - who developed Type II diabetes in their 40s - we're now going to see in people in their late 20s and 30s. If we don't do something about it, our grandchildren are going to go blind in their 20s."

     Diabetes is a fundamental disease that affects the entire colony of cells in a person because it has to do with energy metabolism and the vastly important hormone insulin and its receptor sites. All life is dependent upon basic metabolism, the input of nutrients and removal of wastes. Insulin allows blood sugar (glucose) to be transported into cells so that they can produce energy or store the glucose until it is needed. Insulin binds with receptors on cells like a key would fit into a lock. Once the key insulin has unlocked the door, the glucose can pass from the blood into the cell.  Inside the cell, glucose is either used for energy or stored for future use in the form of glycogen in liver or muscle cells. Most of the food we eat is turned into glucose, which serves as our main source of energy. Insulin is the key for the body's trillions of cells, without it glucose can't get into the cells, so the cells begin to starve. This is all part of a vast network where cells communicate with the intercellular environment through thousands of receptor sites on cell surfaces that respond to thousands of specific molecules (ligands) that bind to these receptors. A receptor that is bound to its activating ligand causes biochemical changes to occur inside the cell. Any problem in this constant communication dynamic between ligands and receptor sites result in disease.

    Diabetes is often conceptualized as a severe imbalance of part of the endocrine system that destroys our ability to metabolize food. The unbalance results in elevated levels of insulin, a lack of insulin, or the cell insulin receptor sites becoming insensitive to insulin. Though we are going to present a non-nutritional and non-lifestyle trigger that sparks a decline into diabetes, the fact remains that the disease has to do with cell nutrition. The essential elements of nutrition will always be important for any lack will make it more difficult for the body to compensate for and correct any systemic problem no matter what the cause.

     Children and adults have been consuming about 80% of their energy intake in the form of highly processed refined carbohydrates which are devoid of minerals and vitamins.  Scientists know that cells cannot convert this caloric energy into cellular energy without nutritional cofactors and this has been a recipe for disaster for a long time. 

Type 1 diabetes, which usually starts in childhood, is a process where the pancreas stops making insulin altogether. It is often called insulin-dependent diabetes. In Type 1 diabetes the cells in the pancreas that make insulin are somehow destroyed, causing a severe lack of insulin. This is thought to be the result of an autoimmune reaction of the body that attacks and destroys the cells in the pancreas. Normal medical explanations for what causes this include infection; exposure to food-borne chemical toxins; and exposure as a very young infant to cow's milk, where an as yet unidentified component of this triggers the autoimmune reaction in the body. The autoimmune process results in the circulation of antibodies that cause beta-cell destruction (the body fighting what it now considers foreign to itself). It is known that certain drugs, such as alloxan, streptozocin, and thiazide diuretics, are toxic to the beta cells of the pancreas and can cause diabetes, but toxic causes have until now been generally ignored. Vaccines have also been implicated in this scenario.

Type 2 diabetes: In 1933 Joslyn, Dublin and Marks published in the American Journal of Medicine a paper titled, "Studies on Diabetes Mellitus" discussing a major epidemic of a disease that looked very much like the Diabetes of the early 1920's only it does not respond to the wonder drug, Insulin. Even worse, sometimes Insulin treatment killed the patient. This disease became known as Insulin Resistant Diabetes because it had the symptoms of Diabetes, but did not respond well to Insulin therapy. Until recently, type 2 diabetes starts in adulthood (and in some teenagers); the body still makes insulin but not enough, or the body can't use what is made with reasonable efficiency. It is often called non-insulin-dependent diabetes. Type 2 diabetes is believed to develop when the receptors on cells in the body that normally respond to the action of insulin fail to be stimulated by it - this is known as insulin resistance. In response to this more insulin is produced and this over-production exhausts the insulin-manufacturing cells. The problem can be one of three: there is simply insufficient insulin available; the insulin that is available may be abnormal and doesn't work properly, or a defect in the insulin receptor sites does not allow insulin to function. 

    The official take on the causes of diabetes is that it is a response to obesity or unhealthy patterns of food intake and energy metabolism. A diet high in refined carbohydrates and sugars, nutrient deficiencies (such as chromium and omega-3 fatty acids), genetic factors, sedentary lifestyle, chronic immune reactivity to dietary antigens and inborn errors of metabolism all are thought to play roles. None of this tells us much about the underlying cause, nothing about how to halt this dramatic rise in human suffering for even with dietary control diabetes is not cured. Though more than 97 million Americans are overweight or obese, diet alone does not explain obesity or diabetes.

     Every cell in the body depends on an adequate supply of glucose for the energy it requires to function properly. For type-2 diabetes mellitus doctors believe that the breakdown in blood sugar regulation results from a sedentary lifestyle, poor diet, and genetic predispositions which all combine to gradually desensitize the body to the actions of insulin, the hormone that transports glucose from the bloodstream into cells. Eventually, it is thought, this malfunction may trigger a vicious cycle of imbalances that promotes further obesity and metabolic imbalances.

     Health officials acknowledge that the inability to maintain normal glucose control can itself lead to obesity, heart disease, hypertension, diabetes, chronic fatigue, accelerated aging, as well as numerous mental and emotional disorders. Doctors routinely tell us that people are developing diabetes because they are fat and that if they fight obesity, they will help prevent diabetes.  Yet the opposite could be true and some people do suspect that people are obese because they are diabetic - not the other way around. The fact that the pancreas secretes excess insulin, the liver manufactures fat from the excess sugar and the adipose cells store excess fat are parts of the diabetic profile suggests obesity is a result not a cause.

     It has long been thought that a diet rich in “empty” sugars will catch our pancreas and adrenal glands in a biochemical see-saw, overworking them and this could weaken the pancreas and result in diabetes. In general though the medical establishment does not recognize the link between sugar consumption and diabetes for if it did there would be warnings against most of the foods found in the supermarket today. Prudence would have us say that sugar over-consumption does not explain the recent rapid rise in diabetes yet it is foolish not to consider it as a background factor. No factor alone can explain anything when it comes to life and health and in a hunt for principal causes this is an important point to retain in mind. We are going to be surprised at what the principle cause actually is, and how other causes tie into the principal one, but there is no denying the facts:

Sugars increase our body's production of adrenaline which puts the body into a state of fight or flight stress, without anything to fight or flee from, except the consumption of sugar. This stress reaction increases the production of both cholesterol and cortisone. White sugar lacks the vitamins and minerals required for its own metabolism. To be metabolized, “empty” sugars must draw on our body's stores of these nutrients. The more sugars you eat, the more vitamins and minerals you need. It can leach B, C, D vitamins, and the following important minerals: calcium, phosphorous, iron, zinc, selenium, and chromium from body tissues. As these are depleted, our body becomes less able to carry out other functions that require minerals and vitamins to be present. Chromium is important here for it is thought to enhance the activity of the hormone insulin. And selenium is useful as a controlling agent for mercury, which attacks insulin and its binding sites. Sir Frederick Banting, the co-discoverer of insulin, noticed in 1929 in Panama that, among sugar plantation owners who ate large amounts of their refined stuff, diabetes was common. Among native cane-cutters, who only got to chew the raw cane, he saw no diabetes. In the 1930s, a research dentist from Cleveland, Ohio, Dr Weston A. Price, traveled around the world and saw that people who live under so-called backward primitive conditions had excellent teeth and wonderful general health.[8] They ate natural, unrefined food from their own locale. As soon as refined and sugared foods were imported, as a result of contact with "civilization", physical degeneration began in a way that was observable within a single generation. Medically, a poison can be any substance ingested which causes disease. White sugar and all the foods that use it can be considered poison, simply because they strip the body of crucial minerals and vitamins. This allows other vastly more toxic poisons to defeat the body’s defenses.

     Problems develop for many people when they regularly eat foods high in refined carbohydrates, such as sugary snacks, pastas, and pizzas. Because these foods break down so rapidly in the gut, they stress the body's use of insulin to control glucose. Combine that situation with low levels of chromium, typical of the American diet, general malnutrition that is creeping into the general public like a permenant fog (nutritional values of food are decreasing rapidly) and a vast increase in chemical toxic stress and we have a recipe for a health disaster.

    To find out the truth about diabetes and why it’s a high speed express running out of control, we have to go further than any of the above explanations. We need to take out our microscopes and investigate what is happening on a biochemical level. We have to forget what everyone says and thinks and all the propaganda we receive from the medical establishment for in this disease, like many others that are going unsolved, direct causes are not being established and thus cures have not been forthcoming. It is no longer easy to trust the medical establishment for they are demonstrating in many areas monumental reasons for that distrust when it comes to chronic diseases like diabetes. Establishing cause leads to rational treatment and drug design and thus to avoiding a great deal of suffering, pain and death.

In 2000-2001, about 82,000 lower-limb amputations
were performed annually among people with diabetes.
In fact, statistics show that every hour, nine people with diabetes
 must have a toe, foot or leg amputated to save their lives
                                                                                               CDC[9]

 

     This is just one gruesome aspect of diabetes, of which there are many. Diabetes has not been cured and insulin is actually highly dangerous, so there is no shortage of motive in searching for a primal precipitating cause that might suggest more effective and safer treatment protocols. Insulin is so prevalent in diabetic treatment but, according to a group of UCLA researchers, heart-suffering diabetics using insulin are 4 times more likely of dying. The figures will be published in the American Heart Journal in January of 2005. According to the researchers, 25-44 percent of heart-suffering people are also diabetic. The nexus between Type 2 diabetes, insulin and heart diseases is renowned, but this is the first time an official document, which will be widely accepted, reports a high death risk due to the use of insulin. The research, carried out on a sample of 554 patients with serious heart problems, proved that the survival rate one year after the diagnosis is 89.7 percent for non diabetic people, 85.5 pct in diabetic cases not treated with insulin, and 62.1 percent for diabetics treated with insulin. Long-term insulin use by patients with Type 2 diabetes is also associated with an increased risk of colon cancer, according to research reported in October of 2004 in the medical journal Gastroenterology. Dr. Yu-Xiao Yang compared insulin use between 125 diabetic patients who developed colon cancer and 1195 similar diabetic subjects who did not. Study results showed that if 100,000 insulin users were followed for 1 year, 197 would develop colon cancer. By contrast, if the same number of non-insulin users were followed, only 124 would develop colon cancer. The risk of colon cancer seems to rise as the duration of insulin use increased. For example, insulin use for 3 to 5 years raised the cancer risk by threefold, whereas use for more than 5 years increased the risk by about fivefold.[10]

    Thus the prognosis is not good for the diabetic patient if you stick with traditional concepts and treatment paths especially that of synthetic insulin, which is about the only insulin a diabetic can buy. These studies are important since they show how crucial it is to keep searching for a cause that will suggest a safer and more effective treatment. Insulin can kill in an instant, hyperglycemia will not. Insulin is a high alert drug because of its ability to throw people into dangerously low blood sugars, and the new insulin analogs are not even insulin but only insulin-like. Diabetics want and need a cure. Prevention would even be better but it has not been forthcoming because no central cause has been identified. At the very least diabetics deserve glucose responsive insulin which is not what they have now. It's been over 80 years since they discovered insulin and in all this time there's no decline in rates of complications. They have not even devised a continuous glucose monitor that will work properly or a closed pump system that works well.

    

Something is not quite right about how doctors conceptualize and treat diabetes. For the children the need to revisit diabetes is vital, for there is an alarming rate of low blood sugars occurring in children at night.  There are alarming reports of doctors now saying that diabetes is a "seizure disorder" because children are having so many seizures from low blood sugars, the kinds of seizures that cause cognitive changes in the brain of developing children. MD's are telling parents to expect these seizures, and that they are part and parcel of diabetes.  This is not quite true but results in putting children on anti-seizure medicine instead of doing something more fundamental to address the low blood sugar.

 

     What we are now going to do is entertain the plausibility that diabetes actually results from poisoning. Though we can spend a lot of time discussing the fact that people can poison themselves with horrible diets, with nutritional deficiencies, that foods full of pesticides, herbicides and preservatives are low grade poisons, that white sugar, for instance, is not a food but another poison that creates all kinds of imbalances, we are going to focus on more powerful poisons. In volume 18 of Clinical Toxicology in 1981[11] there was a write up about cases of suicide attempts using rat poisoning where all four cases showed hyperglycemia and ketosis. The authors concluded that ingestion of rodenticide can cause diabetes mellitus after they noticed that the onset of diabetes mellitus varied within a very short period of time after swallowing of the poison – only 4 to 7 days.

 

     Medical science is just on the brink of discovering how sensitive the insulin receptor sites are to chemical poisoning. Patients treated with the atypical anti-psychotic agents clozapine and olanzapine are showing increased risk for insulin resistance, according to a study published in the January 2005 issue of The Archives of General Psychiatry, one of the JAMA/Archives journals. "Psychiatrists and primary care professionals should be aware that patients treated with clozapine and olanzapine may be at increased risk for insulin resistance, even if not obese. Insulin resistance is associated with hyperlipidemia, hypertension, and cardiovascular disease and over time may increase the risk for diabetes mellitus in vulnerable individuals."[12]

 

    Exactly a year earlier the American Diabetes Association, the American  Psychiatric Association, the North American Association for the Study of Obesity, and the American Association of Clinical Endocrinologists made a similar announcement warning people to be careful to watch for signs they are developing diabetes, obesity or high cholesterol if they are taking Abilify, Clozaril, Geodon, Risperdal, Seroquel or Zyprexa.[13] There is plenty of evidence indicating that chemical poisoning can be placed high on the list of causes for both types of diabetes for again we see that in some cases diabetes symptoms came on suddenly after patients began taking Zyprexa, eased after they went off the drug and returned when they tried the medication again.

 

     The FDA has already requested that Eli Lilly and Company update its product labeling for Zyprexa to include a warning about hyperglycemia and diabetes. Additionally, the British Medical Control Agency and the Japanese Health and Welfare Ministry have both warned about the risk of diabetes in patients who are prescribed Zyprexa. In 2002, a study at Duke University showed a connection between Zyprexa and diabetes. This study documented nearly 300 cases of diabetes in people using Zyprexa. The pharmasceutical industry and particularly Eli Lilly are in serious trouble as lawyers announced the first nationwide class action lawsuit against Eli Lilly and Company on behalf of all persons residing in the United States who used Zyprexa. The lawsuit was filed on April 16, 2004 in the United States District Court for the Eastern District of New York. In December of 2004 a federal judge has told Eli Lilly and Co. to be ready for trial within a year to defend its star drug Zyprexa against charges in federal court that the drug causes diabetes.

     Interesting enough, the CDC offers us a demographic map of diabetes prevalence by state comparing the years 1990, 1996 and 2002 that introduces us to a phenomenon completely overlooked by the medical establishment.

*Includes women with gestational diabetes.
Source: CDC, Behavioral Risk Factor Surveillance System.

     The first thing one should notice about these charts is that there is no significant difference in diet and exercise patterns from state to state to explain the different rates of diabetes we see in the CDC map. What these maps suggest is something entirely different.

 

 

 

 

 

 

    Researchers are baffled by the increased incidence of diabetes in Appalachia. They do not know whether lifestyle, diet, heredity, or a combination of factors is behind the increase. For example, in West Virginia, the only state entirely in Appalachia, 1 in 10 people aged 18 and older was told by a doctor that he or she had diabetes in 2002. Experts give several reasons why diabetes may be widespread in Appalachia, including the heavy and fatty meals the area’s miners, farmers and industrial workers usually eat, but no one is looking into the connection between mercury pollution and diabetes. West Virginia is the nation's second largest coal producer and the state has more than a dozen coal-fired power plants.  West Virginians were warned only recently to limit their consumption of fish in a mercury advisory, which applies to all of the state's waters from its clearest mountain streams to industrial waterways. December’s 2004 mercury advisory, the state's first, was based on a two-year study by West Virginia University.[14] Dr. Robert B. Walker, noting that West Virginia's incidence of diabetes is 41 percent above the national average said, "No disease stresses rural West Virginia families and health providers more than diabetes."[15]

 

Energy Information Administration

Office of Coal, Nuclear, Electric and Alternate Fuels

U.S. Department of Energy

     The incidence of diabetes and end-stage renal disease (ESRD) are closely correlated because diabetes is the most common cause of ESRD, and it accounts for 45% of the new cases of kidney failure in the country. According to the Mid-Atlantic Renal Coalition data for 1998, the ESRD incidence rate for West Virginia was 337.91 per million population. The incidence rate for the United States as a whole for the same year was 310 per million population. West Virginia has one of the highest incidence rates of ESRD of all the states, in large part due to the high percentage of persons with diabetes. In 1999, there were 583 newly diagnosed cases of ESRD with 49% attributed to diabetes as the primary cause. These same kinds of statements can be made for heart disease and other serious complications of health. For instance, the occurrence of pancreatic and liver cancer is greater among diabetes patients. Patients with diabetes mellitus have 3 to 4 times the risk of liver cancer and greater than 2 times the risk of developing pancreatic cancer, compared with non-diabetes patients.[16]

                                           

                1985                                                                                2001

End-Stage Renal Disease

    Geographic variations in adverse health outcomes have long been recognized in the United States, with specific focus on the southeastern region of the country. Cerebrovascular disease mortality rates have identified the Southeast as the "stroke belt" for decades, though rates are also high for other hypertension-related diseases including ischemic heart disease, diabetes, and end-stage renal disease. Although the leading causes of death are similar for the various populations and areas of the United States, significant geographic variation in the level of risks associated with different regions of the country have been documented but what has not been suggested is mercury’s rising role as a central culprit in explaining these geographic differences. It seems like the health establishment is somewhat comatose on the issue of mercury poisoning and chronic disease or just prefers to keep its head stuck in the sand like an ostrich.

     Five southeastern states, Kentucky, West Virginia, Louisiana, North Carolina, and South Carolina are among the 10 states with the highest mortality from ischemic heart disease according to Dr. Daniel Lackland and Dr. Michael Moore. They also detected similar patterns, high mortality rates from diabetes mellitus in the same states, but with diabetes the geographic pattern was somewhat less contiguous. More than 60% of the 16 states in this region were ranked in the upper half for the four health outcomes: Cerebrovascular disease mortality, 88%; ischemic heart disease mortality, 63%; diabetes mellitus mortality, 75%; and incidence of end-stage renal disease, 75%.[17]

     A little further to the north we have bizarre reports that male fish are growing eggs. There is a pollution mystery in West Virginia that is spreading downstream toward Washington. Nine male smallmouth bass, taken from the Potomac about 60 miles from the District, were found to have developed eggs inside their sex organs.[18] Authorities are saying that this is likely related to pollutants that are spewed out by sewage plants, feedlots and factories, and they apparently are able to interfere with the natural hormone systems. "It's certainly something to be concerned about," said Jim Cummins, director of living resources for the Interstate Commission on the Potomac River Basin. "You don't want to see this kind of change in biology."

    Actually up and down the eastern part of the United States we find higher levels of mercury than just about anywhere else. The above maps and demographics set the stage for a serious proposal that mercury poisoning is at the heart of the modern plague of diabetes, and thus also of heart and kidney disease, strokes, blindness, and other common complications of diabetes.

    These maps make a strong and important graphic point but they do not reflect on the greatest source of mercury pollution, the toxic waste dumps that are installed in peoples’ mouths by dentists. We ingest the greatest volume of mercury on a daily basis directly from our dental amalgam fillings. These maps are illustrating another dimension of mercury toxicity which is growing and finally threatening but, according to the World Health Organization, dental amalgam is ten times more threatening in terms of volume of mercury absorbed. We can reasonably assume that dental amalgam is evenly distributed across the United States so it seems like environmental factors are gaining potency adding to the badly leaking toxic mercury waste sites in peoples’ mouths. Eating mercury polluted fish is an obvious danger that is threatening portentously. And so is taking a growing list of pharmaceutical drugs that together with mercury seem to be delivering knockout punches to the glucose control mechanisms of the body.   

     There are trends that seem to correlate with the skyrocketing diabetes pandemic. First, these past decades has seen a rapid rise in the prevalence of mercury (most toxic poison out there) in the environment, in peoples’ teeth, and since around 1990, more used in vaccines hidden behind the name of thimerosal. The upward curve of age related onset of diabetes can clearly be correlated with the years of continued mercury exposure through constant vapor inhalation from dental amalgams, which through the decades eventually overwhelms all capacities of the body to eliminate and sustain mercury’s onslaught.

             Proportion of female population with confirmed diabetes in 2002.

 

 

     The most recent epidemic in child onset diabetes has been correlated with the increase in vaccines which deliver more and more toxic chemicals into babies’ bodies. No one in mainstream medicine is taking into account the intensifying increase in background mercury contamination of fish, water, air, soil and foods or calculating the hundreds of trillions of mercury atoms and molecules being absorbed directly each day, day after day, year after year through having gram weight quantities placed directly in the mouth. Before we dive into the molecular science of mercury toxicity it might be helpful to read some unusual case studies. Though not usually used as proof or evidence of anything, case studies bring us face to face with individual realities and what our intellectual arguments and studies eventually lead us to in the end. The rejection of antidotal stories is telling and is a way the medical establishment has been able to stone wall common sense. Though we are not presenting these two short case stories as proof of anything, we would say that ignoring such evidence is abusive. A good doctor does not have to hear many times that something is dangerous to his patients to be careful.

 

“When I was six years old I remember being fascinated with the thermometers my mother used when I was sick. The thermometer had that silver liquid that flowed in it, and I did not know anything about it being poisonous. But I do remember breaking one on purpose to investigate the mercury, and playing with it as a child. I never told my mother because of the fact I broke one of her thermometers, but if it is true it causes major damage for insulin receptors and production I may have doomed myself as a child by playing with the mess in the thermometer my mother used that I broke. I was six, and that was the year I came down with insulin dependent diabetes.” DJ

 

     Finally, after decades of use, the danger of mercury thermometers is known. Several states and even countries are passing laws banning their use. Acute mercury poisoning can kill. Falling short of that, a mercury spill from a thermometer can cause serious harm. It is standard medical establishment behavior though to argue about how that harm manifests. What disease a person will suffer from, short of dying, is unknown until it manifests in an individual. In the case of DJ the obvious fallout from the mercury spill (inhalation of mercury vapor) was destruction of the beta cells in the pancreas.  

 

“I was diagnosed juvenile diabetic at 14 years, am 49 now, male, married with 3 kids. In all these years nobody has ever asked me to explain my problems that deal with mercury. The eyes roll and head shake sums it up most of the time I bring it up.  When I was 5 years old a silver amalgam filling was put in an incisor tooth. That baby tooth fell out when I was about 10 years old.  The next day when I woke up I was shocked at how well I felt and I didn't have a clue as to why. Later that morning I saw that tooth and the silver filling and I thought, would anybody put poison in a little kid’s mouth. I thought no, and forgot about it. Soon my speech straightened out, I could think clearly, I wasn’t dizzy all the time, I quit urinating a lot  and I felt I wasn't holding it all the time. About 6 months after this filling fell out another bigger silver amalgam was put in. Within a few days I was urinating at school and I thought to myself, its back. I knew there was something bad in silver amalgams but didn’t want to believe it, so I forgot about it.  When I was 13 years old, 9 silver amalgams were put in my mouth. Something bad happened when the last filling was put in my mouth, on the walk home I thought  to myself, I'm screwed and life as I knew it is over.  I thought nobody would believe me and nobody did. About a year later I was a diagnosed juvenile diabetic.” Terry

 

    Enzymes are proteins, and like all proteins they consist of chains of amino acids.  These chains have to be faulted in a specific way to give the enzyme its activity. In many enzymes, the structure of the enzyme is ensured by cross-bonding of the amino-acid chains. These cross-bonds consist of double sulfur bonds. Sulfur-bridges are covalent S-S bonds between two cysteine amino acids, which tend to be quite strong. These sulfur bonds are damaged when poisonous substances that are not naturally present have been added to the local environment. Mercury binds to the -SH (sulfhydryl) groups, resulting in inactivation of sulfur and blocking of enzyme functions while producing sulfur metabolites with high toxicity that the body has difficulty dealing with.  Sulfur is essential in enzymes, hormones, nerve tissue, and red blood cells. These sulfur bonds are crucial to human biology.

 

     Metals such as iron, mercury, arsenic, lead and possibly aluminum may play a role in the actual destruction of beta cells through stimulating an auto-immune reaction to them after they have bonded to these cells in the pancreas. What we will focus on here though is the fact that insulin has three sulfur-containing cross-linkages and the insulin receptor has a tyrosine kinase-containing sulfur bond, which are the preferred targets for binding by both mercury and lead. Should mercury attach to one of these three sulfur bonds it will interfere with the normal biological function of the insulin molecule. In reality there is no should about it, the average adult inhales many trillions of mercury atoms a day from a mouth full of amalgam, fish provide trillions more, the air more, and in children, vaccines provide one day surges of vast trillions of mercury molecules in the form of ethyl-mercury, which is vastly more toxic than metallic mercury. Insulin molecules are directly assaulted as are insulin receptor sites.

 

 

 

 

Thiol poisons, especially mercury and its compounds, reacting with
 SH groups of proteins lead to the lowered activity of various enzymes
 containing sulfhydryl groups. This produces a series of disruptions in
 the functional activity of many organs and tissues of the organism.
                                                                 Professor I.M. Trakhtenberg[19]
                                                                                                     Russia

     Each insulin molecule consists of precisely 2 peptide chains (A and B) bound together by sulfa bonds at the A7-B7 Cysteine site and at the A20-B19 Cysteine site and there is an additional Cysteine sulfa bond at the A6-A11. All insulin molecules consist of this two chain structure, with an A chain of 21 amino acids and a B chain of 30 amino acids, for a total of 51 amino acid molecules bound by 3 sulfa bonds.   Mercury, in its various forms, has a great attraction to the sulfhydryls or thiols. A thiol is any organic compound containing a univalent radical called a sulfhydryl and identified by the symbol -SH (sulfur-hydrogen).

     Various molecules or atoms will affect the rate of an enzyme catalyzed reaction by binding to the 
enzyme. Some bind at the same site as the substrate (the active site) and prevent the substrate from 
binding. Others bind at sites on the enzyme remote from the active site and affect activity by modifying 
the shape of the enzyme. Many of these molecules reduce the activity of the enzyme and are referred to 
as inhibitors. Mercury is the most potent enzyme inhibitor that exists; it is in a class of its own and 
well deserves its title as the most toxic non-radioactive element. It is because mercury and lead attach 
themselves at these highly vulnerable junctures of proteins that they find their great capacity to provoke 
biochemical shifts and then morphological changes in the body. Transsulfuration pathways in the body 
are fundamental for life. When mercury blocks thiol groups cellular proteins lose their reactive properties, 
lose their ability to carry out their routine function. 

Because glycemic regulation is one of the body’s most central homeostatic mechanisms,
mercury’s attack is most problematic, even at low concentrations, and indicates
that it is playing a great role in the dramatic rise in diabetes.

     Equally vulnerable to mercury’s ruin are the receptor tyrosine kinases (RTKs) which are glycoproteins that transduce insulin’s extracellular signal to the cytoplasm of the cell. The effects of insulin are mediated by the insulin receptor (specific RTK for insulin). When insulin binds with its receptor, the receptor activates and recruits a whole chain of downstream signaling processes. The RTK insulin receptor is comprised of two extracellular alpha chains disulfide-linked to two membrane-spanning beta chains. The three dimensional crystal structure of insulin-like growth factor 1 (IGF1) receptor provides a clue to the complete vulnerability of humans when it comes to mercury’s destructive power that can lead to diabetes. The molecular structure of both IGF1 and the RTK insulin receptor sites are rich in cysteines and as such we find an array of disulfide-linked modules that mercury penetrates. Published studies from Northeastern University with thimerosal show that it inhibits the ability of insulin-like growth factor-1 (IGF-1) to activate the enzyme methionine synthase. Decreased activity of IGF-1 signaling is associated with type 1 diabetes, particularly a failure of signaling in insulin-secreting beta islet cells. A single preliminary experiment at the Joslin Diabetes Clinic showed that thimerosal inhibits an early step in the signaling pathway. Studies at Northeastern also provided evidence that Cu2+ stimulates the IGF-1 signaling pathway, and it appears that thimerosal is also interfering with this normal activity. Some doctors have speculated about vaccines being responsible for the increases we are seeing in children’s diabetes but now it is becoming clearer that children’s systems are under a broad mercury attack with each source, type of mercury, and mode and timing of contamination setting different stages for different pathologies.

 

It is the inability to see the effects of chronic,

low level toxicities on human health that has been,
 and remains, our greatest failing as intelligent beings.
                                                                        Dr. Boyd Haley                                                                                                                 

     The general model of insulin activity indicates that one insulin molecule engages the cystein-rich domain of the receptor, touching down on both sides of protein chain that are separated by the disulfide bond. If the geometry of the receptor has been changed by mercury the message that insulin has arrived to give the cell is not received. Mercury is an inhibitor capable of interfering with PTK catalytic activity exactly because it is collapsing/damaging these sulfur-containing cross-linkages which changes the geometry of both insulin receptor and insulin itself.

 

1. Insulin binds to and activates the insulin receptor. 
2. Insulin receptor activation stimulates the movement of chromium into the cell. 
3. Chromium binds to a peptide known as Apo-LMWCr* (Apo-LC). *LMWCr = low-molecular weight chromium-binding substance
4. Functional LMWCr (LC) binds to the insulin receptor and enhances its activity.[20]

     Adult onset (type 2) diabetes develops when glucose homeostatic regulation breaks down. 
Most type-2 diabetics produce enough insulin but have developed resistance to normal insulin 
action in target tissues and now we have an explanation why. When we look at the fact that peripheral 
cells each have approximately 3,500 receptor sites, (other cells have less) that there are 70 trillion cells 
in the body; and thousands of trillions of atoms an average person with a mouth full a amalgam might 
ingest each day, we can see why diabetes tends under most conditions, even with mercury directly 
assaulting these sulfur bonds, to be a slow onset disease and that’s why the prevalence increases with 
age. When the available sites on the cells drop into the 1,200 to 1,500 ranges the cell and entire insulin 
hormone system run into serious trouble. When it comes to children and early onset diabetes thimerosal 
is one likely villain. Thimerosal is ethyl-mercury mixed with aluminum in the vaccine and if anyone ever 
wanted to discover the ultimate elixir of death, they probably found it. Injecting millions of children with 
150,000 trillion atoms of supercharged death particles (50 micrograms in two thimerosal containing 
vaccines) is high insanity, surely the ultimate treason against a generation of children. 

      With only 70 trillion cells in the body of an adult, a six month old child of few kilos could be vaccinated
and have atom sized meteors aimed at each and every cell of their body. For an adult it would be two 
mercury atoms for each cell of the body. For a child its many more since vaccines are not administered 
adjusted according to a person’s weight. The simple math indicates that there is easily enough mercury to 
attack every cell in a child’s body. If a child’s cells are already weakened from previous vaccines, mercury 
entering in their food, air and water, from mother’s blood and mercury teeth fillings, and weakened by 
thousands of other chemicals they are being exposed to early in life; if they are thus weakened a new assault 
of aluminum supercharged thimerosal atoms can be the end for a child, or the beginning of something that 
will only get worse over time – diabetes. 

Vaccines are the largest cause of insulin dependent diabetes in children.
                                                                                    Dr. J. Barthelow Classen

     Dr. J. Barthelow Classen published, in the Journal of Pediatric Endocrinology and Metabolism (2003), his research into the links between vaccine and insulin-dependent diabetes. Classen's research indicates that vaccinations can cause autoimmune diseases and he cites research from New Zealand indicating that the incidence of diabetes there has increased by 50 percent since 1988 when hepatitis B was given to New Zealand children under 16. He also cited a rapid rise in the incidence of childhood diabetes since the introduction of the HiB vaccine--50 cases of diabetes per 100,000 vaccinated children. According to Classen, vaccines in general can cause interferon release, which can in turn induce human diabetes. “Our results conclusively prove there is a causal relationship between immunization schedules and diabetes,” states Dr. Classen. A mother from Wisconsin with fraternal twins that have both autism and type 1 diabetes was concerned about the possibility of connection between these two disorders. Mercury poisoning from vaccines and other sources offers us a common denominator. Mercury is a poison whose avenue of assault is diverse attacking nervous system tissues and insulin receptor sites while provoking auto-immune responses simultaneously with ease.

     The results of all the above are threatening devastation around the world. At the very end of 2004 the 
New Zealand Herald reported that “nearly half the adult population surveyed for a study on the East Coast 
have been found to be insulin resistant, putting them at greater risk of developing type 2 diabetes.”[21]

Mercury interacts with sulfhydryl groups and disulfide bonds,
 as a result of which specific membrane transport is blocked
 and selective permeability of the membrane is altered.

    Thus insulin's ability to lower blood sugar is compromised. Mercury is causing chronic hyperinsulinemia because insulin resistance causes serum hyperglycemia and intra-cellular hypoglycemia (since glucose cannot get into cells) and the cells perceive this state as starvation.[22] As a consequence, blood glucose levels become so elevated that the glucose "spills over" into the urine, and is converted into triglycerides (fat), which contribute to cardiovascular illness. Despite these high blood glucose levels, cells "starve" since insulin-stimulated glucose entry into cells is impaired, which leads to an acid condition, ketosis. To compensate, the body releases glugcagon, cortisol, and catecholamines which further raise blood sugar levels. More insulin is pumped out resulting in hyperinsulinemia and this creates a cascading biological disaster of triglyceride and cholesterol excess, hypertension, inflammatory cytokine release and further endocrine dysfunction.[23] [24]

 

Peripheral neuropathy is a common condition that can cause
 numbness and tingling. It can be caused by diabetes or
 overexposure to toxic chemicals, such as mercury or lead.
                                                                                         Park Nicollet Institute

     It is through mercury’s attack on these sulfide bonds that mercury is able to change the biological properties of proteins and change important physiological functions. Full recognition of the poisonous strength of mercury comes through a focused study of the series of physiological processes that are affected when SH groups are disturbed. It is vitally important that doctors know that mercury can change the chemical structure of a molecule without producing an evident toxic effect. Thus doctors are deceived when diabetics demonstrate major tissue and organ damage, aging, uremia, blindness (retinopathy), nerve damage, (neuropathy), cardiovascular disease, dementia, and renal damage (nephropathy).[25] Thinking that high levels of glucose in the blood and tissues is damaging and disrupting healthy cell function throughout the body (via structural mutations of proteins, a process known as glycation or glycosylation which creates deformed, dirty, toxic encrusted proteins that create all the problems mentioned in the preceding sentence) doctors and research scientists lose site that mercury is acting in the place of first cause creating the high levels of glucose through its interference in insulin and insulin receptor sites. All these glycated proteins are creating 50 times the amount of free radicals than non-glycated proteins and from here it’s a downhill losing battle. Atherogenesis including inflammation, impaired vascular dilation, hypertension, increased clotting, and through reduced clearance of lipids,[26] excessive cross-linking of collagen causes arterial walls to thicken. The body is literally slowly poisoning itself but it’s not doing that because of a genetic defect, chance of fate or because of lack of exercise and improper diet, though most of these factors make matters worse. It’s doing it because it is being poisoned by mercury on a molecular level. Through many years