please find our latest media release (below) and two important new factsheets (attached). Apologies for cross-postings.
Yours Sincerely Elizabeth O'Brien, President, The LEAD Group Inc. PO Box 161 Summer Hill NSW 2130 Australia Ph +61 2 9716 0132 www.lead.org.au
Working to eliminate lead poisoning in Australia & further afield by the year 2012 & to protect the environment from lead. The LEAD Group Inc. ABN 25 819 463 114
Monday 17th August 2009 - FOR IMMEDIATE RELEASE
NHMRC going nowhere on Australian blood lead levels
A goal of less than 10 micrograms of lead per 100 millilitres of blood (10 ug/dL) for all Australians thats the same as they announced in 1993, said Elizabeth OBrien of The LEAD Group, referring to the National Health and Medical Research Councils latest public statement and information paper (6/8/09) on blood lead levels. Babies and baby boomers are still not protected. Doctors will be no better-informed and no action is recommended by NHMRC. Wow!
In 1993, said Ms OBrien, NHMRCs goal was for a blood lead level of less than 10 ug/dL, and the target to be achieved by 1998, was for all Australians with the exception of those occupationally exposed to lead, to have a blood lead level less than 15ug/dL. This target has not yet been achieved. The NHMRC rescinded its 1993 policy in 2005. Now it has resurfaced.
However, unlike 1993, this time the NHMRC has not set any target dates at all, requires no government action and barely mentions potential adult fatalities from lead, said Ms OBrien.
The NHMRC does not describe the most critical research during the last 16 years, except to downplay it, which indicates harmful effects on childrens brain development and behaviour, and increased risk of heart attacks and strokes in adults, at levels as low as 2 ug/dL.
The latest statement on lead by the NHMRC could have set the goal to be lower than 5 ug/dL and the target to be lower than 10 ug/dL by 2012, recommended national 5 yearly blood lead studies, as well as research into prevention of deaths from the lead that all adults store in their bones and which slowly re-enters their blood stream as they age. That would be worth reading.
Goal. Target. It sounds like a word game. The difference is, a goal is what you want to achieve for everyone to be below a certain level. A target is a level you aim to achieve, within a stated time frame. Just having a goal on its own is expressing a wish, but if theres no target then theres no series of steps any agency or GP or cardiologist is recommended to take for achieving that goal. Theres also no way of checking whether youre making any progress.
The way to check for progress on something like this is to do blood lead studies, of all ages. Not the whole population - sample groups, as advised by a statistician; without compromises. That way you get trustworthy results and can work out where the worst cases are and why.
Theres not been such a survey in Australia, so theres no information about how common an elevated blood lead level is say, over 2 ug/dL it could be the majority of Australians. Without such data, and without adequate information for doctors on the recently discovered adverse health effects of blood lead levels even below 10 ug/dL, there is little to motivate them to order blood lead tests. Patients have to ask! Many will die never knowing lead killed them.
apologies for cross-postings but an Australian caller to our lead information service does not yet have email and wants to find a woman who has experienced chelation treatment for lead poisoning, to talk on the phone with her lead poisoned daughter (who also doesn't yet have email and lives in NSW, Australia). Andrew Hobday, who is a founding member of these egroups, has kindly offered to talk on the phone to such people and I've given his phone number to the caller, but because the daughter has questions related to women's health, she would also like to talk to a woman. Could you please email me at egroup@... with your phone number if you are willing?
Cheers
Yours Sincerely Elizabeth O'Brien, Manager, Global Lead Advice & Support Service (GLASS) run by The LEAD Group Inc. PO Box 161 Summer Hill NSW 2130 Australia Ph +61 2 9716 0132 Freecall 1800 626086 www.lead.org.au
we have a new member who is writing a novel with historical reference to workers in a white lead paint factory, and I wrote the following email to the author and then realized you may all be interested in my email and the associated library articles.
Please feel free to write and tell me your actual name in case it is not Yoger Bo - I had to put something into the Yahoo form when I added you to the LeadWorkers egroup.
Good luck with your novel!
Please don't hesitate to send queries to either egroup but especially to LeadWorkers egroup.
You will find that one LPAE member has written a whole website - http://www.bellsystemleadpoisoning.com/ - case history of his father who was lead poisoned through soldering, and one member of LeadWorkers is the lead poisoned painter son of a lead poisoned painter.
You may also find the following articles from our library useful:
"Poorly Controlled Hypertension in a Painter with Chronic Lead Toxicity"] at
"Use Of Lead In Paint In NSW - When And Where It Was Used" by John Hartley, BCCA (The Blast Cleaning and Coating Association of Australia) [attached, filename: BCCA Use of lead in paint updated 20060131.doc]
"Lead Less Toxic to the Well-Read: Good reading ability may help protect the brain from damage linked to toxic lead, a new study [in lead smelter workers in New Brunswick, Canada] shows" [NO LONGER AT ORIGINAL WEBSITE BUT SIMILAR ARTICLE FOUND AT http://health.usnews.com/usnews/health/healthday/070730/lead-less-toxic-to-the-well-read.htm ] (attached, filename: FORBES dot com - Lead Less Toxic to the Well-read smelter worker 20070730.doc)
"Introduction: Industry's Child" from the book "Deceit and Denial: The Deadly Politics of Industrial Pollution" by Rosner, David and Markowitz, Gerald, AT www.ucpress.edu/books/pages/9844/9844.intro.html
"The Lead Industry and Child Lead Poisoning" by Richard Rabin AT
"The Harmful Cerussa (White Lead), That Most Noxious Thing" [QUOTED IN the book: "Measuring Lead Exposure in Infants, Children, and Other Sensitive Populations" originally cited by Major, 1945, p. 312] AT http://books.nap.edu/openbook.php?record_id=2232&page=13 (attached, Filename: Nikander - The harmful cerussa, white lead, that most noxious thing QUOTE 2nd Century BC.doc]
Yours Sincerely Elizabeth O'Brien, Winner of the United Nations Assoc'n of Australia (UNAA) World Environment Day (WED) Award for Outstanding Service to the Environment. Manager, Global Lead Advice & Support Service (GLASS) run by The LEAD Group Inc. PO Box 161 Summer Hill NSW 2130 Australia Ph +61 2 9716 0132
Hi new here and looking for insights and support. I tend to
research things and then decide what to do next.
I am over 50, f, and live in the Pacific Northwest US. I was raised
in the NW, but had spent about 20 years in the Midwest US working
before moving back. I have also lived in the southwest.. I was
forced out of work for health reasons and am now retired officially.
I have always been chemically sensitive I suspect. I showed my first
signs at age 10. At age 30 it manifested into food allergies. I had
hepatitis first as a toddler and a few other rounds of liver problems
since. The cause is unknown (isn't that typical doctor talk).
A year ago I got really sick. I was grasping at straws looking for
answers. We had acquired a used refrigerator and looking back, I got
sicker after working on cleaning the dusty junk under it on the coils
and fan; it has now been replaced. The road in front had weed killers
put on it in a routine spraying the month before I started cleaning
the fridge and that got me sick too. Even though I am to be notified
in advance I wasn't but I would have gotten sick anyhow.
In looking for answers, I had a DDI hair test run in October 2007. It
showed elevated arsenic and had the markers for adrenal failure (high
K and Na and low Ca and Mg). The primary care person I see had no
idea how to interpret it (she didn't order it either) and I got some
insight on a yahoo group and ended up buying Cutler's Hair
Interpretation book. The elevated arsenic made sense if the previous
owner of the refrigerator had put rat or mouse poison on it and I got
it on me somehow.
So I have spent this last year working on adrenal issues and dealing
with what I assumed was arsenic poisoning. I have attacked this with
supplements and I have to admit when I feel worse, I have a hard time
keeping them up. When I push things, I get more headaches, and have
strong smelling urine.
So fast forward to now. I ran another hair test a couple weeks ago.
This one still shows arsenic in the yellow zone, but now antimony is
also yellow. I also have a 4 fold increase in lead (still green) and
see ups and downs in other toxic elements. The adrenal issues are
better but still there. The potassium is still sky high, but not as
high. If it helps I can post the hair tests on photobucket.
I was looking for mercury as I still have my metal fillings. The
counting rules show "passing" in other words none of them are met
but I understand the presence of these other toxins can mask that.
I don't know if the antimony is new or old stuff being excreted.
Where is the antimony coming from? I wish I knew. The bed is 10
years old but I added what was then a year old memory foam topper
(this was after I got sick). I had worked about 40 years ago for a
short time in electronics in plants with printed circuit boards and
soldering operations. I wasn't actively involved as I did clerical
and quality control work.
Our water supply is "safe" with levels < 0.005 - up to 6 is allowed on
the last test. I don't know about where we had lived before haven't
researched that yet. I do read about antimony leeching into plastic
water bottles and well I do drink bottled water. At home I tend to
drink steam distilled. I do have a new distiller.
I do know they paved the roads recently here (first coat was July 17th
in front) and that work with asphalt and oil was pretty toxic and I
had been doing better before they started doing that again this
summer. .
My symptoms include some brain fog. My reflexes were shot a year ago
but are better. I have metallic tastes, gum irritations (but dentist
says things are okay) and distorted sense of smell. I had some pain
in the teeth with metal fillings during all of this and had rotten
tastes in my mouth around the same time. I have rounds of emotional
issues. I have ear pains too and hearing is super sensitive according
to my spouse. The distorted smells drive me crazy and I suspect I am
detoxing stuff and smelling that (and it is causing the gum sensations
but I really don't know).
The policy is The LEAD Group’s contribution to the NHMRC’s (National Health and Medical Research Council) recent Canberra meeting to develop such a policy for Australia.
“The problem of lead poisoning goes beyond Australia,” says Elizabeth O’Brien, President of The LEAD Group. “And what goes around comes around – lead-contaminated food, toys and other products being a case in point.”
“Australia’s lead (and lead-containing coal) is exported to the world. It’s therefore appropriate and a moral imperative, that The LEAD Group “export” a policy, via our website, for preventing lead poisoning and contamination.”
The model policy outlines the steps national health ministers need to take to cover the three phases of preventing lead poisoning: primary, secondary and tertiary prevention.
“A critical mass of research exists for national governments to consult, in formulating the specific details of their own policy,” said Ms O’Brien, “but, we also recommend they ask the World Health Organisation to carry out research in tertiary prevention – that is, preventing the lead already in a person’s body from causing further harm. We all carry a burden of lead because of “the mistake of the 20th century” – adding lead to petrol.”
“Research is needed on whether treatment or other interventions prevent any of the associated adverse effects that are linked with lead: infertility, sub-optimal foetal and childhood development, later schizophrenia, Alzheimer’s, and hypertension to name a few” she said.
“Rapid industrialization, by India and China in particular, means the adoption by those countries, of a national policy for preventing lead poisoning is vital for the health of their populations. Because of the high prevalence of lead poisoning in China, The LEAD Group has just web-published our first non-English language factsheet, in Mandarin.” See 铅,衰老和死亡- “Lead, Ageing and Death.”
“The LEAD Group’s continued existence and growth will help promote adoption, by the countries of the world, of the model health policy for the achievement of our ultimate goal – global lead poisoning elimination and the protection of the environment from lead.”
Contact: Elizabeth O’Brien, The LEAD Group - +61 2 97160132, mobile +61 431184933###
It's little wonder that I was attracted to singing in the Beethoven Society
of Australia Choir!
Elizabeth O'Brien
----- Original Message -----
From: "Rebekah Waechter"
To: <leadnet@...>
Sent: Wednesday, August 29, 2007 5:22 AM
Subject: [Leadnet] Expert: Beethoven inadvertently poisoned by doctor
VIENNA, Austria (AP) -- Did someone kill Beethoven? A Viennese pathologist
claims the composer's physician did -- inadvertently overdosing him with
lead
in a case of a cure that went wrong.
http://www.cnn.com/2007/SHOWBIZ/Music/08/28/whokilledbeethoven.ap/index.html
Expert: Beethoven inadvertently poisoned by doctor
a.. Story Highlights
b.. Beethoven was treated for fluid in the abdomen
c.. Doctor treated him by puncturing abdomen, covering with
lead-containing balm
d.. Lead in Beethoven's body could have also been from other sources
e.. Beethoven died at age 57 in 1827
VIENNA, Austria (AP) -- Did someone kill Beethoven? A Viennese pathologist
claims the composer's physician did -- inadvertently overdosing him with
lead in a case of a cure that went wrong.
For the great Ludwig van Beethoven, the treatment may have been worse than
the disease.
Other researchers are not convinced, but there is no controversy about one
fact: The master had been a very sick man years before his death in 1827.
Previous research determined that Beethoven had suffered from lead
poisoning, first detecting toxic levels of the metal in his hair and then,
two years ago, in bone fragments. Those findings strengthened the belief
that lead poisoning may have contributed -- and ultimately led -- to his
death at age 57.
But Viennese forensic expert Christian Reiter claims to know more after
months of painstaking work applying CSI-like methods to strands of
Beethoven's hair.
He says his analysis, published last week in the Beethoven Journal, shows
that in the final months of the composer's life, lead concentrations in his
body spiked every time he was treated by his doctor, Andreas Wawruch, for
fluid inside the abdomen. Those lethal doses permeated Beethoven's ailing
liver, ultimately killing him, Reiter told The Associated Press.
"His death was due to the treatments by Dr. Wawruch," said Reiter, head of
the Department of Forensic Medicine at Vienna's Medical University.
"Although you cannot blame Dr. Wawruch -- how was he to know that Beethoven
already had a serious liver ailment?"
Nobody did back then.
Only through an autopsy after the composer's death in the Austrian capital
on March 26, 1827, were doctors able to establish that Beethoven suffered
from cirrhosis of the liver as well as edemas of the abdomen. Reiter says
that in attempts to ease the composer's suffering, Wawruch repeatedly
punctured the abdominal cavity -- and then sealed the wound with a
lead-laced poultice.
Although lead's toxicity was known even then, the doses contained in a
treatment balm "were not poisonous enough to kill someone if he would have
been healthy," Reiter said. "But what Dr. Wawruch clearly did not know that
his treatment was attacking an already sick liver, killing that organ."
Even before the edemas developed, Wawruch noted in his diary that he treated
an outbreak of pneumonia months before Beethoven's death with salts
containing lead, which aggravated what researchers believe was an existing
case of lead poisoning.
But, said Reiter, it was the repeated doses of the lead-containing cream,
administered by Wawruch in the last weeks of Beethoven's life, that did in
the composer.
Analysis of several hair strands showed "several peaks where the
concentration of lead rose pretty massively" on the four occasions between
December 5, 1826, and February 27, 1827, when Beethoven himself documented
that he had been treated by Wawruch for the edema, said Reiter. "Every time
when his abdomen was punctured ... we have an increase of the concentration
of lead in the hair."
Such claims intrigue others who have researched the issue.
"His data strongly suggests that Beethoven was subjected to significant lead
exposures over the last 111 days of his life and that this lead may have
been in the very medicines applied by his doctor," said Bill Walsh, who led
the team at the U.S. Department of Energy's Argonne National Laboratory
outside Chicago that found large amounts of lead in Beethoven's bone
fragments. That research two years ago confirmed the cause of years of
debilitating disease that likely led to his death -- but did not tie his
demise to Wawruch.
"I believe that Beethoven's death may have been caused by this application
of lead-containing medicines to an already severely lead-poisoned man,"
Walsh said.
Still, he added, samples from hair analysis are not normally considered as
reliable as from bone, which showed high levels of lead concentration over
years, instead of months.
With hair, "you have the issue of contamination from outside material,
shampoos, residues, weathering problems. The membranes on the outside of the
hair tend to deteriorate," he said, suggesting more research is needed on
the exact composition of the medications given Beethoven in his last months
of his life.
As for what caused the poisoning even before Wawruch's treatments, some say
it was the lead-laced wine Beethoven drank. Others speculate that as a young
man he drank water with high concentrations of lead at a spa.
"We still don't know the ultimate cause," Reiter said. "But he was a very
sick man -- for years before his death."
The Beethoven Journal is published by the Ira F. Brilliant Center for
Beethoven Studies at San Jose State University in California.
I trust that this study (please see article in email below) will also look into exposure to lead fumes for the circuit board workers, as lead is now regarded as a probable human carcinogen and IBM should have blood lead records going back further than any other biological monitoring for an occupataional exposure. Can someone from NIOSH please find out for us and let ABLES members know - I will then pass the news on to the LeadWorkers egroup.
Cheers
Elizabeth O'Brien
Moderator, LeadWorkers egroup
Manager, Global Lead Advice & Support Service
PO Box 161 Summer Hill NSW 2130 Australia Ph +61 2 9716 0132
Subject: [csda] IBM cancer study gets federal push (NY)
IBM cancer study gets federal push $3.2M in funding almost '100 percent certain' By Tom Wilber Binghamton Press & Sun-Bulletin, NY
Federal scientists will have money to proceed with a monumental study of cancer rates among 28,000 IBM-Endicott employees that will advance science about illness and chemical exposure, U.S. Rep. Maurice Hinchey said Thursday.
Hinchey, a member of the House Appropriations Committee, said he has secured funding necessary for the National Institute of Occupational Safety & Health to begin the $3.2 million study in the next fiscal year, which begins in October.
It would be the first comprehensive government study of cancer rates among employees in the circuit board manufacturing industry, and is intended to address widespread questions about whether they bore a disproportionately high cancer risk.
"The funding has been secured. It is in the budget," said Hinchey, D-Hurley. "NIOSH has given us assurances they will begin the study next fiscal year." The funding was included in a clause in a bill passed by the House that would make the study a priority for NIOSH in 2008.
Meanwhile, IBM pledged to cooperate with the study.
"We will pass along any information (to NIOSH) that is lawfully acceptable," IBM spokesman Ari Fishkind said Thursday afternoon. "We have cooperated with NIOSH in the past and that will always be the case."
The study would be based largely on records dating to the early 1960s that document the IBM work force at the sprawling Endicott facility, now owned by Huron Real Estate Associates. They would be cross-referenced with cancer and death records kept by state and federal government agencies, so researchers will be able to tell if a person who worked for the company for a given period developed cancer any time after that.
The study also would tap IBM's industrial hygiene records to track what chemicals were used, where, and when in an attempt to characterize likely exposure scenarios for workers in various departments.
The study would be a significant contribution to worldwide occupational safety, including countries that use chemicals and processes being phased out of this country, said Richard Clapp, professor of environmental health at Boston University and a national authority on chemical exposure.
"It's an understudied industry," he said. "This is a big step forward."
Fred Blosser, a spokesman for NIOSH in Washington, on Thursday said the agency could not comment on funding for the agency until the budget is finalized. In the meantime, he added, agency officials would "watch with interest" the progress of the legislation containing the clause for the Centers for Disease Control and Prevention to proceed with the IBM study. NIOSH is a branch of the CDC.
"We appreciate the congressman's interest and we will follow the progress of the bill," Blosser said.
Lynne Pinkerton, a scientist with NIOSH, said in a proposal earlier this year that IBM was willing to contribute personnel records, but not eager to share industrial hygiene information. The agency has the authority to subpoena the records. If that becomes necessary, a legal battle could delay the study.
Hinchey has advocated tougher state and federal TCE regulations after the chemical was found wafting from polluted sites into buildings throughout the Southern Tier in 2003. To date, more than 500 properties have been affected in Endicott, Hillcrest, Binghamton, Vestal and Norwich.
A study in one of those areas, south and southwest of the former IBM plant on North Street, Endicott, showed residents had disproportionately high rates of testicular and kidney cancer, and heart defects.
The IBM study would pertain to exposure to many kinds of chemicals, including TCE.
About 40 people, including advocates, residents and state and local elected officials, applauded Hinchey as he walked into the village rotunda for the press conference Thursday afternoon, and again when it was over.
Rick White, a spokesman for Alliance@IBM, a labor advocacy group, said Thursday he has growing confidence IBM will cooperate with the study.
* The NIOSH proposal to study IBM-Endicott workers' cancer rates is available at the George F. Johnson Library in Endicott or online at: www.cdc.gov/niosh/review/public/103 ..
* Comments on the study will be accepted until Aug. 20 and can be submitted online to: www.cdc.gov/niosh/review/public/103 or mailed to: NIOSH Docket Office, 4676 Columbia Parkway, MS C-34, Cincinnati, OH 45226.
The August 2007 issue of Environmental Health Perspectives is now available. Below are highlights of a few papers that may be of particular interest to the news media.
Stress Modifies Lead Effect on Hypertension Lead exposure and psychological stress have been independently associated with hypertension in various populations, and animal studies suggest that when they co-occur, their effects may be exacerbated. Researchers examined whether psychological stress modifies the impact of cumulative lead exposure (measured as bone lead levels) on hypertension and blood pressure in Boston (Massachusetts) area community-exposed men participating in the Normative Aging Study. These are the first analyses to look at interactive effects of stress and lead on hypertension in humans. The results suggest that the effect of lead on hypertension is most pronounced among highly stressed individuals, independent of demographic and behavioral risk factors. View Article <http://www.ehponline.org/docs/2007/10002/abstract.html> Childhood Blood Lead Levels and Educational Outcomes Childhood lead poisoning remains a critical environmental health concern. To determine whether lead levels in early childhood are related to educational achievement as measured by performance on end-of-grade (EOG) testing, researchers linked test data for 4th-grade students to blood lead surveillance data for seven North Carolina counties. They found that discernible impact of blood lead levels on EOG testing is demonstrated for early childhood blood lead levels as low as 2 g/dL; a blood lead level of 5 g/dL is associated with a decline in reading and mathematics scores. Early childhood lead exposures appear to have more impact on performance on the reading than on the mathematics portions of the tests. View Article <http://www.ehponline.org/docs/2007/9994/abstract.html>
Media may also be interested in EHPs news articles. Among this months articles is a discussion of how environmental factors interact with mental illness (see Environmental Connections: A Deeper Look into Mental Illness <http://www.ehponline.org/docs/2007/115-8/focus-abs.html> ). Another feature examines the growing worldwide trend against public smoking (see A Change in the Air: Smoking Bans Gain Momentum Worldwide <http://www.ehponline.org/docs/2007/115-8/spheres-abs.html> ).
*You received this e-mail as a member of the working media and a registrant on our press page. EHP is published by the National Institute of Environmental Health Sciences, part of the U.S. Department of Health and Human Services. If you no longer wish to get our press releases, simply let us know by responding to this e-mail.
MONDAY, July 30 (HealthDay News) -- Good reading ability may help protect the brain from damage linked to toxic lead, a new study shows.
Lead was found to be 2.5 times more likely to have negative effects on the brains of adults with limited reading ability than on the brains of good readers, the researchers report in the July 31 issue of Neurology.
However, reading ability did not protect individuals' motor skills from the toxic effects of lead.
A team at the Center for Occupational and Environmental Neurology, in Baltimore, studied the effects of lead exposure on 112 lead smelter workers in New Brunswick, Canada. The workers took several thinking and motor-speed tests as well as a measure of their reading ability.
The researchers then calculated working lifetime lead exposure from historic blood lead levels obtained by the smelter. The workers were divided into groups with "high cognitive reserve" -- defined as a reading level of 12th grade or higher -- and "low cognitive reserve," a reading level of 11th grade or lower.
Cognitive reserve refers to the mental abilities, such as reading ability, that are generally not affected by lead exposure in adulthood. They act as a measure of the brain's ability to maintain function despite damage.
The results: "Even though the two groups had similar lead exposure, the cognitive effects of lead were 2.5 times greater in workers with low reading ability," study author Dr. Margit L. Bleecker said in a prepared statement. "In contrast, the effect of lead on motor speed was comparable in both groups as cognitive reserve does not apply to motor speed," she said.
"This suggests that high cognitive reserve has a protective effect that allowed these workers to maintain their functioning, even though lead affected their nervous system as shown by its effect on their motor skills," Bleecker added.
How might reading protect the brain? According to the researchers, an increased number of cortical synapses in larger brains might provide more brain capacity, the option to use alternative brain circuits if some are damaged, and the ability to process tasks more efficiently.
Subject: [LeadWorkers] Australian TV show Catalyst will hopefully mention lead poisoning today & tomorrow 26/7 etc
Hi all,
I noticed in an ad for Catalyst on Australian Broadcasting Commission (ABC) Channel 2 will screen a story about Californian Condors and since they were nearly wiped out by lead poisoning and the remaining few have been undergoing chelation therapy, I figured that these important details will be mentioned in the story (though they are not mentioned in the promo below).
The California Condor flies again, Brain Overload, The Sun in Stereo, Meet "Palaeo-Artist" Dr. Anne Musser
Thursday, 26 July 2007
The California Condor flies again
Imagine being born in captivity, spending your whole life constrained, and then one day you're set loose in the big wild world.
Well, California Condor Number 79 will find out what that's like, when she's given her first taste of freedom.
From the magnificent mountains of the USA's west coast, Dr Jonica Newby reports on the moving tale of how a small band of conservationists dreamed of seeing this giant of the Pleistocene flying once more above the Californian coastline.
This is a story of ancient mystery and intrigue, of courage and fear, and above all, of an incredible battle to bring a prehistoric species back to life.
Reporter Dr. Jonica Newby Producer Paul Faint Researcher: Maria Ceballos
I noticed in an ad for Catalyst on Australian Broadcasting Commission (ABC) Channel 2 will screen a story about Californian Condors and since they were nearly wiped out by lead poisoning and the remaining few have been undergoing chelation therapy, I figured that these important details will be mentioned in the story (though they are not mentioned in the promo below).
The California Condor flies again, Brain Overload, The Sun in Stereo, Meet Palaeo-Artist Dr. Anne Musser
Thursday, 26 July 2007
The California Condor flies again
Imagine being born in captivity, spending your whole life constrained, and then one day youre set loose in the big wild world.
Well, California Condor Number 79 will find out what thats like, when shes given her first taste of freedom.
From the magnificent mountains of the USAs west coast, Dr Jonica Newby reports on the moving tale of how a small band of conservationists dreamed of seeing this giant of the Pleistocene flying once more above the Californian coastline.
This is a story of ancient mystery and intrigue, of courage and fear, and above all, of an incredible battle to bring a prehistoric species back to life.
Reporter Dr. Jonica Newby Producer Paul Faint Researcher: Maria Ceballos
> > > Liz. > > If the study is right I should have died years ago,I bought and sold > more then > > 500,000 lbs. of lead a year. I touched every bit of this lead, > And I have a wife. > > I also worked at agas station in the sixties. I was tested for > lead every year, > > My numbers were at 5 or below. Also,I played with mercy in high > school > > labs. > > > Yours > > Mike Circle > > > > ----- Original Message ----- > From: egroup@... > To: LeadWorkers@yahoogroups.com > > Sent: Thursday, July 12, 2007 2:12 > AM > Subject: Re: [LeadWorkers] Is full lead > paint removal the best public health policy? Article on lead & brain > cancer > > > > So what you're saying then Mike, is that, if children > are lucky enough to live with a person who is professionally trained in > OH&S to deal with lead safely and if those parentsapply that > training to their renovation techniques and their parenting, and if they have > their kids routinely blood lead tested when they are little so they know that > their methods really are lead safe, then repainting over the top of the lead > paint is fine? I think that just leaves the vast majority of the population > for whom a public health policy of full paint removal is > essential! > I just received another scarey article on the impacts of > lead poisoning in workers, which adds to the argument for much better OH&S > training regarding lead. > Link to article abstract on lead and > brain cancer. > http://www.rxpgnews.com/cancer/brain/article_4897.shtml > The article begins: > People who are routinely exposed to lead on the > job are 50 percent more likely to die from brain cancer than people who are > not exposed, according to a University of Rochester Medical Center > study. > Regards > Elizabeth O'Brien > > ----- Original Message ----- > From: michael > circle > To: LeadWorkers@yahoogroups.com > > Sent: Thursday, July 12, 2007 11:10 AM > Subject: Re: [LeadWorkers] Is full lead paint removal the best > public health policy? > > > Elizabeth > > I have rehab. Over (5) homes that had lead based paint on them, The > homes > > were tested and found that if the old paint is cleaned and prepped > > > the right way the homes were safe for children including my own. > > I find if I feed my children they do not eat the paint off the walls and > shills, > > there fore they are safe from lead poisoning. > > > > Mike Circle > > > > > > > > > > > >
Lead must be ingested or inhaled..you can't get lead poisoning by touching it. Paint on homes and walls are eaten by children (chips) or they fall in the soil and contaminate the
ground water etc. Jennifer
Jennifer Winter---
CoreComm Webmail
http://www.core.com
Subject: Re: [LeadWorkers] Is full lead paint removal the best public health policy? Article on lead & brain cancer
So what you're saying then Mike, is that, if children are lucky enough to live with a person who is professionally trained in OH&S to deal with lead safely and if those parents apply that training to their renovation techniques and their parenting, and if they have their kids routinely blood lead tested when they are little so they know that their methods really are lead safe, then repainting over the top of the lead paint is fine? I think that just leaves the vast majority of the population for whom a public health policy of full paint removal is essential!
I just received another scarey article on the impacts of lead poisoning in workers, which adds to the argument for much better OH&S training regarding lead.
Link to article abstract on lead and brain cancer.
People who are routinely exposed to lead on the job are 50 percent more likely to die from brain cancer than people who are not exposed, according to a University of Rochester Medical Center study.
So what you're saying then Mike, is that, if children are lucky enough to live with a person who is professionally trained in OH&S to deal with lead safely and if those parents apply that training to their renovation techniques and their parenting, and if they have their kids routinely blood lead tested when they are little so they know that their methods really are lead safe, then repainting over the top of the lead paint is fine? I think that just leaves the vast majority of the population for whom a public health policy of full paint removal is essential!
I just received another scarey article on the impacts of lead poisoning in workers, which adds to the argument for much better OH&S training regarding lead.
Link to article abstract on lead and brain cancer.
People who are routinely exposed to lead on the job are 50 percent more likely to die from brain cancer than people who are not exposed, according to a University of Rochester Medical Center study.
Subject: [LeadWorkers] Is full lead paint removal the best public health policy?
Dear LeadWorkers Egroup,
the question came up today that there is an inconsistency between the Australian Standard on Lead Paint Management AS4361.2 and the policy of the Global Lead Advice and Support Service (GLASS) in terms of whether to recommend full lead paint removal or simply repainting after lead-safe preparation of the old paint. The Australian Standard recommends against full lead paint removal but GLASS is totally for full lead paint removal. I thought it would be good if people are interested in this inconsistency, that they read the article below that influenced me to set the GLASS policy. And if you are as convinced by it as I was, then you are encouraged to write to SAI Global (the new name for Standards Australia) and ask them to review the Standards in line with good public health policy (ie full lead paint removal).
Please let me know what you think.
Elizabeth O'Brien, Manager, GLASS
The New England Journal of Medicine -- May 10, 2001 -- Vol. 344, No. 19
Primary Prevention of Childhood Lead Poisoning -- The Only Solution[LID 5453]
[Source: First one hundred word extract at http://content.nejm.org/cgi/content/short/344/19/1470 but requires free registration at http://content.nejm.org/cgi/content/full/344/19/1470 for the full (2 page) article]
Lead poisoning in children was first recognized in its severe acute form, known as lead encephalopathy.
This condition is characterized by seizures, coma, and -- not infrequently -- death, and it is associated with severe neurologic sequelae in survivors. Although lead encephalopathy has become rare in the past 15 to 20 years, the dangers of clinically asymptomatic lead poisoning in children have become increasingly clear.
Longitudinal studies of development from birth to adolescence show that irreversible cognitive damage can occur with blood lead levels considerably lower than those typically associated with overt symptoms. Recognition of this problem has led to routine screening programs and clear guidelines for the management of severe lead poisoning (defined by whole-blood lead levels of 45 g per deciliter or more). In contrast, the appropriate management of moderate lead poisoning (defined by whole-blood lead levels of 20 to 44 g per deciliter) remains uncertain.
In this issue of the Journal, Rogan et al. report the results of a multicenter, randomized trial of an oral lead-chelating agent, succimer, in children whose blood lead levels were 20 to 44 g per deciliter. The performance of these children on several cognitive assessments was below average -- an observation consistent with previous reports of deficits in academic achievement, abstract thinking, attention span, conceptual reasoning, and visuospatial perception in children with moderately high blood lead levels. Although chelation therapy lowered blood lead levels, it had no effect on any of the several neurobehavioral and cognitive measures used in the study. The study suggests that even with succimer therapy, the neurocognitive effects of chronically elevated blood lead levels and total-body lead burden are irreversible. Such irreversibility of developmental deficits due to neurotoxicity has been documented previously in children who did not receive chelation therapy.
Chelation therapy was initially introduced to manage severe, and frequently acute, lead poisoning that would most likely result in death or severe, overt neurologic sequelae in survivors. In affected children, whose blood lead levels were often greater than 100 g per deciliter, chelating agents rapidly lowered blood lead levels by brisk diuresis and stopped the progression of lead poisoning to the point of frank encephalopathy. This treatment saved lives but did not eliminate the neurologic consequences, which were permanent. Very rarely, if ever, does chelation therapy for such severe lead poisoning reverse or prevent the signs and symptoms of lead-induced neurotoxicity. Rather, the benefit of chelation therapy for children with severe acute or chronic lead poisoning is defined in relation to the terrible prognosis for children who are not so treated.
Chelation therapy is now used routinely in children who have blood lead levels of 45 g per deciliter or more, with the goal of preventing neurologic deterioration, lead encephalopathy, and death. However, there have been no clear guidelines from the Centers for Disease Control and Prevention or other advisory bodies regarding therapy for children with blood lead levels of 20 to 44 g per deciliter, who were represented by the participants in the present study.
The study by Rogan et al. used the most promising chelating agent, succimer, and what was considered to be a highly effective dosing regimen. We believe it unlikely that any other study design, study population, chelating agent, or dosing regimen would produce a materially different result. Neurocognitive tests assessed measures characteristically affected by lead toxicity. The study population accurately reflected, both demographically and socioeconomically, the children at highest risk in urban communities in the United States.
The lack of efficacy of succimer for the prevention of neurotoxic harm due to moderate blood lead levels in the children in the study by Rogan et al. and the limited efficacy of chelation therapy in cases of severe lead poisoning clearly cast doubt on the value of public health programs that rely primarily on treatment after lead poisoning has occurred. Rogan et al. emphasize the importance of the primary prevention of lead poisoning, which is the only satisfactory solution to this devastating problem. The predominant source of toxic exposure to lead for children in urban areas is lead paint, although some incremental but far less substantial toxicity may be due to other sources, such as tap water contaminated by lead pipes.
For the primary prevention of lead poisoning from paint, we recommend permanent abatement -- that is, the complete removal or replacement of lead paint before a child lives in a home. In contrast, "interim" measures, which were introduced for the short-term reduction of hazards associated with lead paint and which involve scraping and painting over deteriorated surfaces and controlling household dust, have been claimed by some to save substantial cost; however, there is no evidence of savings in terms of net benefit over cost in the long-term prevention of childhood lead poisoning. Lead-painted surfaces in good condition rarely remain so. What was once intact lead-based paint is the source of all lead-bearing dust and paint chips. Therefore, it is the presence of lead paint on surfaces that defines the hazard, not the condition of surfaces containing lead paint.
Although succimer therapy resulted in lower blood lead levels, its failure to reverse neurocognitive deficits in the study by Rogan et al. confirms the need for collective and concerted efforts to prevent lead poisoning in children.
the question came up today that there is an inconsistency between the Australian Standard on Lead Paint Management AS4361.2 and the policy of the Global Lead Advice and Support Service (GLASS) in terms of whether to recommend full lead paint removal or simply repainting after lead-safe preparation of the old paint. The Australian Standard recommends against full lead paint removal but GLASS is totally for full lead paint removal. I thought it would be good if people are interested in this inconsistency, that they read the article below that influenced me to set the GLASS policy. And if you are as convinced by it as I was, then you are encouraged to write to SAI Global (the new name for Standards Australia) and ask them to review the Standards in line with good public health policy (ie full lead paint removal).
Please let me know what you think.
Elizabeth O'Brien, Manager, GLASS
The New England Journal of Medicine -- May 10, 2001 -- Vol. 344, No. 19
Primary Prevention of Childhood Lead Poisoning -- The Only Solution[LID 5453]
[Source: First one hundred word extract at http://content.nejm.org/cgi/content/short/344/19/1470 but requires free registration at http://content.nejm.org/cgi/content/full/344/19/1470 for the full (2 page) article]
Lead poisoning in children was first recognized in its severe acute form, known as lead encephalopathy.
This condition is characterized by seizures, coma, and -- not infrequently -- death, and it is associated with severe neurologic sequelae in survivors. Although lead encephalopathy has become rare in the past 15 to 20 years, the dangers of clinically asymptomatic lead poisoning in children have become increasingly clear.
Longitudinal studies of development from birth to adolescence show that irreversible cognitive damage can occur with blood lead levels considerably lower than those typically associated with overt symptoms. Recognition of this problem has led to routine screening programs and clear guidelines for the management of severe lead poisoning (defined by whole-blood lead levels of 45 g per deciliter or more). In contrast, the appropriate management of moderate lead poisoning (defined by whole-blood lead levels of 20 to 44 g per deciliter) remains uncertain.
In this issue of the Journal, Rogan et al. report the results of a multicenter, randomized trial of an oral lead-chelating agent, succimer, in children whose blood lead levels were 20 to 44 g per deciliter. The performance of these children on several cognitive assessments was below average -- an observation consistent with previous reports of deficits in academic achievement, abstract thinking, attention span, conceptual reasoning, and visuospatial perception in children with moderately high blood lead levels. Although chelation therapy lowered blood lead levels, it had no effect on any of the several neurobehavioral and cognitive measures used in the study. The study suggests that even with succimer therapy, the neurocognitive effects of chronically elevated blood lead levels and total-body lead burden are irreversible. Such irreversibility of developmental deficits due to neurotoxicity has been documented previously in children who did not receive chelation therapy.
Chelation therapy was initially introduced to manage severe, and frequently acute, lead poisoning that would most likely result in death or severe, overt neurologic sequelae in survivors. In affected children, whose blood lead levels were often greater than 100 g per deciliter, chelating agents rapidly lowered blood lead levels by brisk diuresis and stopped the progression of lead poisoning to the point of frank encephalopathy. This treatment saved lives but did not eliminate the neurologic consequences, which were permanent. Very rarely, if ever, does chelation therapy for such severe lead poisoning reverse or prevent the signs and symptoms of lead-induced neurotoxicity. Rather, the benefit of chelation therapy for children with severe acute or chronic lead poisoning is defined in relation to the terrible prognosis for children who are not so treated.
Chelation therapy is now used routinely in children who have blood lead levels of 45 g per deciliter or more, with the goal of preventing neurologic deterioration, lead encephalopathy, and death. However, there have been no clear guidelines from the Centers for Disease Control and Prevention or other advisory bodies regarding therapy for children with blood lead levels of 20 to 44 g per deciliter, who were represented by the participants in the present study.
The study by Rogan et al. used the most promising chelating agent, succimer, and what was considered to be a highly effective dosing regimen. We believe it unlikely that any other study design, study population, chelating agent, or dosing regimen would produce a materially different result. Neurocognitive tests assessed measures characteristically affected by lead toxicity. The study population accurately reflected, both demographically and socioeconomically, the children at highest risk in urban communities in the United States.
The lack of efficacy of succimer for the prevention of neurotoxic harm due to moderate blood lead levels in the children in the study by Rogan et al. and the limited efficacy of chelation therapy in cases of severe lead poisoning clearly cast doubt on the value of public health programs that rely primarily on treatment after lead poisoning has occurred. Rogan et al. emphasize the importance of the primary prevention of lead poisoning, which is the only satisfactory solution to this devastating problem. The predominant source of toxic exposure to lead for children in urban areas is lead paint, although some incremental but far less substantial toxicity may be due to other sources, such as tap water contaminated by lead pipes.
For the primary prevention of lead poisoning from paint, we recommend permanent abatement -- that is, the complete removal or replacement of lead paint before a child lives in a home. In contrast, "interim" measures, which were introduced for the short-term reduction of hazards associated with lead paint and which involve scraping and painting over deteriorated surfaces and controlling household dust, have been claimed by some to save substantial cost; however, there is no evidence of savings in terms of net benefit over cost in the long-term prevention of childhood lead poisoning. Lead-painted surfaces in good condition rarely remain so. What was once intact lead-based paint is the source of all lead-bearing dust and paint chips. Therefore, it is the presence of lead paint on surfaces that defines the hazard, not the condition of surfaces containing lead paint.
Although succimer therapy resulted in lower blood lead levels, its failure to reverse neurocognitive deficits in the study by Rogan et al. confirms the need for collective and concerted efforts to prevent lead poisoning in children.
Interested people who are willing to put a Lead Recycling Plant to
recycle Lead acid battery scrap we can design and manufacture plants
of international standards our manufacturing base is in India and we
have the competence and capacity to design small, medium and mega size
Lead plants with sophisticated and proven technology.
Drop in email at metchem_engg@...
Regards
Tarum
Hello Damien,
From my experience, and several others who I have come across with
high lead exposure, CFS (Chronic Fatigue Syndrome) symptoms have
definitely been present.
...And probably the most persistent.
Andrew
***************************************************
--- In LeadWorkers@yahoogroups.com, "damienhorler" <wacket@...> wrote:
>
> hi leadworkers
> my name is damien I am a new member and need some info.
> I have been diagnosed with ME/CFS 3 years ago and believe exposure
> to lead a few years before may have some thing to do with it.
> I have just recently had a iv chelation challenge where they test
> the urine which picked up lead.
> I was a builder and at one job I was using second weatherboards
> that had lead paint on them, I used a heat gun that bubbles the
paint
> so you can scrap it off. I did have a proper mask on but do
remember
> smelling it through the mask occassionally. I was also cutting the
boards
> as well.so I need info
> I need to find a doctor who knows about this stuff
> I need to know the best way to chelate the lead.
> I need to know if people have cfs type symptoms with lead poisoning
> thanks damien
>
> Damien: Did you get a lead blood level test? There is also a blood test for lead in tissue known as a Zinc Protporphin (sp) test. The symtoms of CFS and lead poisoning are very similar and may very well be your problem. The bad news is that after chelation therapy
there is no cure or treatment for lead accumulated in your
body except time. The lead accumulates in the bone and
stays for many years. Good luck...Jennifer
>
> hi leadworkers
> my name is damien I am a new member and need some info.
> I have been diagnosed with ME/CFS 3 years ago and believe exposure
> to lead a few years before may have some thing to do with it.
> I have just recently had a iv chelation challenge where they test
> the urine which picked up lead.
> I was a builder and at one job I was using second weatherboards
> that had lead paint on them, I used a heat gun that bubbles the paint
> so you can scrap it off. I did have a proper mask on but do remember
> smelling it through the mask occassionally. I was also cutting the boards
> as well.so I need info
> I need to find a doctor who knows about this stuff
> I need to know the best way to chelate the lead.
> I need to know if people have cfs type symptoms with lead poisoning
> thanks damien
>
>
>
>
>
>
>
>
>
>
>
>
>
>
>
>
Jennifer Winter---
CoreComm Webmail
http://www.core.com
hi leadworkers
my name is damien I am a new member and need some info.
I have been diagnosed with ME/CFS 3 years ago and believe exposure
to lead a few years before may have some thing to do with it.
I have just recently had a iv chelation challenge where they test
the urine which picked up lead.
I was a builder and at one job I was using second weatherboards
that had lead paint on them, I used a heat gun that bubbles the paint
so you can scrap it off. I did have a proper mask on but do remember
smelling it through the mask occassionally. I was also cutting the boards
as well.so I need info
I need to find a doctor who knows about this stuff
I need to know the best way to chelate the lead.
I need to know if people have cfs type symptoms with lead poisoning
thanks damien
[For those people receiving this who are not in the Lead Poisoned Adults
Egroup (LPAE), please refer to the email below which inspired this
investigation of possible answers, which itself raises important questions
for everyone receiving this email.]
I am starting to believe that the best answer to the perennial question of
whether the damage lead has done to an individual is permanent or whether
some symptoms are reversible, is to ask another question: what is your known
or likely cumulative blood lead index plus what are the other health risks
you have acquired by dint of your family medical history, your upbringing
and lifetime exposures to other harbingers of ill-health?
To explain the concept of your known or likely cumulative blood lead index,
there are precious few references because the science of predicting health
effects in an individual is dependent on the science of epidemiology which
has a hard enough time determining risks across a whole population of people
whose blood lead levels (or bone lead levels) are measured either once or
over a period of time (as in longitudinal studies). The unfortunate fact of
the matter is that, especially for people outside the USA, known cumulative
blood lead indices are extremely rare and would normally only be able to be
calculated for lead exposed workers IF the lead-exposed worker is fortunate
enough to work for a company that actually complies with OH&S regulations
which demand workers blood lead monitoring data be kept AND the company is
prepared to release the results to the worker even if the worker is retired
or has left.
The cumulative blood lead index is measured in ug-years/dL. A ug year/dL in
lead exposure is one microgram of lead per decilitre of blood for a period
of one year. The recent mini-monograph in the March 2007 Environmental
Health Perspectives included the article "Adult Lead Exposure: Time for
Change" by Brian S. Schwartz and Howard Hu (see
http://www.ehponline.org/members/2006/9782/9782.pdf) and concludes (in
part):
"The growing body of scientific evidence suggests that occupational
standards should limit recent dose to prevent the acute effects of lead and
separately limit cumulative dose to prevent the chronic effects of lead."
"We would favor limits that keep blood lead levels < 20 ug/dL to prevent the
acute effects of recent dose. For the prevention of the chronic health
effects of cumulative dose, the available evidence suggests (snip)
maintaining the cumulative blood lead index below approximately 200-400
ug-years/dL (equivalent to an average blood lead level of 20 ug/dL for 10-20
years or of 10 ug/dL for 20-40 years).
The question which arises is: which among the Australian lead mining
companies and state and federal OH&S regulatory bodies in the US and
Australia are going to incorporate this new recommendation into their policy
or regulations?
I am very keen to hear from anyone who has the power to do so.
Regards
Yours Sincerely
Elizabeth O'Brien, Manager, Global Lead Advice & Support Service
PO Box 161 Summer Hill NSW 2130 Australia
Ph +61 2 9716 0132
www.lead.org.au
----- Original Message -----
From: "brindle222"
To: <LPAE@yahoogroups.com>
Sent: Sunday, March 11, 2007 9:40 AM
Subject: [LPAE] Newest member
In June of 2006 I was diagnosed with lead and mercury poisoning, by a
provoked 24 urine test. Since September I have had 21 EDTA one hour
chelation IV's. I do feel better, but I can't find any answers as to
whether there is permanent damage and/or will I ever feel normal again,
if I can remember what normal is. I have been sick since I turned 60
and I will be 68 in a few days. In 2003 I had to retire, because I no
longer could funtion more than 2 days a week. My lead count was 48 in
June and it is now down to 8. The mercury seems to have cleared. Any
information would be greatly appreciated.
Yahoo! Groups Links
Please find below the more recent email that I intended to attach to the email I sent you all on 3rd March 2007 with the following note:
This is a groundbreaking recommendation (see extract below) that needs to be advocated for at every opportunity.
Can you write to your politicians to ask them to review current policy that allows workers to be subjected to a blood lead level of 35-50 ug/dL or more before action is taken, and does very little to even measure blood lead levels of adults in the general population or high-risk groups like renovators, shooters, leadlighters and other hobbyists?
Or suggest other ways to change the mindset about what's an acceptable blood lead level for adults.
Kind regards
Elizabeth O'Brien
----- Original Message ----- From: "Kelly O'Grady" <lead@...> To: <leadnet@...> Sent: Friday, March 02, 2007 3:33 AM Subject: [Leadnet] EHP Adult Lead Poisoning Monograph Series
New articles in March 2007 Environmental Health Perspectives:
Conclusions: At exposure levels encountered after environmental exposure, associations with biomarkers of cumulative dose (mainly lead in tibia) were stronger and more consistent than associations with blood lead levels. Similarly, in studies of former workers with past occupational lead exposure, associations were also stronger and more consistent with cumulative dose than with recent dose (in blood) . In contrast, studies of currently exposed workers generally found associations that were more apparent with blood lead levels ; we speculate that the acute effects of high, recent dose may mask the chronic effects of cumulative dose. There is moderate evidence for an association between psychiatric symptoms and lead dose but only at high levels of current occupational lead exposure or with cumulative dose in environmentally exposed adults
Ana Navas-Acien,1 Eliseo Guallar,2,3 Ellen K. Silbergeld,1 and Stephen J. Rothenberg4
Lead Exposure and Cardiovascular Disease?"A Systematic Review
Excerpt:
Conclusions: We conclude that the evidence is sufficient to infer a causal relationship of lead exposure with hypertension. We conclude that the evidence is suggestive but not sufficient to infer a causal relationship of lead exposure with clinical cardiovascular outcomes. There is also suggestive but insufficient evidence to infer a causal relationship of lead exposure with heart rate variability.
Public Health Implications: These findings have immediate public health implications. Current occupational safety standards for blood lead must be lowered and a criterion for screening elevated lead exposure needs to be established in adults. Risk assessment and economic analyses of lead exposure impact must include the cardiovascular effects of lead. Finally, regulatory and public health interventions must be developed and implemented to further prevent and reduce lead exposure.
Michael J. Kosnett,1 Richard P. Wedeen,2 Stephen J. Rothenberg,3,4 Karen L. Hipkins,5 Barbara L. Materna,6 Brian S. Schwartz,7,8 Howard Hu,9 and Alan Woolf10
Recommendations for Medical Management of Adult Lead Exposure
Environmental Health Perspectives Volume 115, Number 3, March 2007
Excerpt:
Based on this literature, and our collective experience in evaluating lead-exposed adults, we recommend that individuals be removed from occupational lead exposure if a single blood lead concentration exceeds 30 µg/dL or if two successive blood lead concentrations measured over a 4-week interval are ? 20 µg/dL. Removal of individuals from lead exposure should be considered to avoid long-term risk to health if exposure control measures over an extended period do not decrease blood lead concentrations to < 10 µg/dL or if selected medical conditions exist that would increase the risk of continued exposure. Recommended medical surveillance for all lead-exposed workers should include quarterly blood lead measurements for individuals with blood lead concentrations between 10 and 19 µg/dL, and semiannual blood lead measurements when sustained blood lead concentrations are < 10 µg/dL. It is advisable for pregnant women to avoid occupational or avocational lead exposure that would result in blood lead concentrations > 5 µg/dL.
Howard Hu, Regina Shih, Stephen Rothenberg, and Brian S. Schwartz
The Epidemiology of Lead Toxicity in Adults: Measuring Dose and Consideration of Other Methodologic Issues
Environmental Health Perspectives Volume 115, Number 3, March 2007
Excerpt:
The strong associations of cumulative lead dose with race/ethnicity and socioeconomic status raises methodologic concerns. Factors that in the past were simply termed ?oconfounding variables? are now more carefully evaluated as potential mediators (i.e., in the biological causal pathway), moderators (i.e.,risk modifiers), direct causes, or otherwise parts of complex causal pathways (Kraemer et al. 2001). It is now understood that such complex causal pathways also apply to lead exposure and chronic disease, including cognitive dysfunction, hypertension, and renal dysfunction. These pathways can include connections between individual-level indicators (e.g., age, sex, race/ethnicity, socioeconomic status); behavioral risk factors; biological factors (e.g., genetics); social factors (e.g., social capital, social cohesion); lead dose (i.e., both recent and cumulative); health conditions (e.g., diabetes, heart disease, hypertension); and other biological markers predictive of disease (e.g., homocysteine levels) that may be thought of as either outcomes by themselves or as intermediate pathological states that result in other conditions (e.g., renal dysfunction, cognitive declines)
Environmental Health Perspectives Volume 115, Number 3, March 2007
Excerpt:
The lead standards of the U.S. Occupational Safety and Health Administration are woefully out of date given the growing evidence of the health effects of lead at levels of exposure previously thought to be safe, particularly newly recognized persistent or progressive effects of cumulative dose. The growing body of scientific evidence suggests that occupational standards should limit recent dose to prevent the acute effects of lead and separately limit cumulative dose to prevent the chronic effects of lead. We hope this mini-monograph will motivate renewed discussion of ways to protect lead-exposed adults in the United States and around the world.
The First Six Years (formerly known as Lead Environmental Awareness and Detection l.e.a.d.) is a grass roots organization whose primary mandate is the identifcation and prevention of pediatric lead exposure in Renfrew County which occurs from residential sources: i.e. lead (paint, paint dust, water, soil, other). We promote the development of optimal social and physical environmental conditions for healthy productive children through blood lead screening; environmental monitoring and surveillance; and public and professional education.
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We would favor limits that keep blood lead levels < 20 ug/dL to prevent the acute effects of recent dose. For the prevention of the chronic health effects of cumulative dose, the available evidence suggests that tibia lead levels should not be allowed to exceed 15 ug lead/g bone mineral; this could also be achieved by maintaining the cumulative blood lead index below approximately 200-400 ug-years/dL (equivalent to an average blood lead level of 20 ug/dL for 10-20 years or of 10 ug/dL for 20-40 years). Unfortunately, other scientists and public health professionals made similar recommendations more than 15 years ago (Landrigan et al. 1990; Silbergeld et al. 1991), and little has resulted. We hope this mini-monograph will have a larger impact on policy.
This is a groundbreaking recommendation (see extract below) that needs to be advocated for at every opportunity.
Can you write to your politicians to ask them to review current policy that allows workers to be subjected to a blood lead level of 35-50 ug/dL or more before action is taken, and does very little to even measure blood lead levels of adults in the general population or high-risk groups like renovators, shooters, leadlighters and other hobbyists?
Or suggest other ways to change the mindset about what's an acceptable blood lead level for adults.
Kind regards
Elizabeth O'Brien
----- Original Message ----- From: Hipkins, Karen (DHS-OHB) To: ABLES@... Sent: Wednesday, December 27, 2006 6:22 AM Subject: EHP 5 part minimonograph series on adult lead exposure
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Lead Exposure and Cardiovascular Disease - a S... Navas-Acien A, et al.
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We would favor limits that keep blood lead levels < 20 ug/dL to prevent the acute effects of recent dose. For the prevention of the chronic health effects of cumulative dose, the available evidence suggests that tibia lead levels should not be allowed to exceed 15 ug lead/g bone mineral; this could also be achieved by maintaining the cumulative blood lead index below approximately 200-400 ug-years/dL (equivalent to an average blood lead level of 20 ug/dL for 10-20 years or of 10 ug/dL for 20-40 years). Unfortunately, other scientists and public health professionals made similar recommendations more than 15 years ago (Landrigan et al. 1990; Silbergeld et al. 1991), and little has resulted. We hope this mini-monograph will have a larger impact on policy.
The United Nations and everyone else seem perfectly happy to take UNTIL 2010 to remove lead from all petrol in the remaining 50 or so countries where it's still sold for road-use!! And to me - that's just not darn-well soon enough!! Please read the appeal below and figure out SOON whether you can help. It's also on our home page www.lead.org.au but you have to SCROLL DOWN to see it!
Cheers
Elizabeth O'Brien
ph +612 9716 0014
Urgent Appeal for Funding!
Your Assistance Is Needed To Help Achieve A Global Ban On Leaded Petrol.
The LEAD Group makes an urgent appeal to our web readers for donations to send our representative and member of the Partnership for Cleaner Fuels and Vehicles (PCFV), Elizabeth OBrien, to the United Nations Environment Programmes 5th Global Partnership Meeting. The event will be held in Quito, Ecuador, 15‑16 February 2007.
The meeting will be an important milestone in the work of the Partnership for Clean Fuels and Vehicles (PCFV) and will map out the strategies need to achieve the phase-out of leaded petrol around the world.
In June 2004 Elizabeth was honoured with the Award for Outstanding Service to the Environment by the United Nations Association of Australia. Elizabeth attended the 3rd Global meeting in Delhi, India, in 2004.
No other Australian representative has attended any other of the annual meetings, yet The LEAD Group believes that it is Australian lead that is used to make the leaded petrol for all remaining 80 or so countries where leaded petrol is sold. Therefore we feel it is important that Australia is represented at this meeting.
The LEAD Group seeks a quick global end to the use of leaded gasoline and believes that it is the most important public health goal of the last century. The PCFV appears to be the only process that can achieve this global ban within an acceptable time frame.
The approximate costs of attendance to the Global Meeting include return flights from Sydney to Quito (from $AUD 2,850- not including government charges and taxes) the hotel accommodation ($USD 75 per night, for a regular room at Swissotel, for one week), the cost of meals (lunch will be catered for on the 2 days of the meeting) and transfers to and from the airport in Quito.
Any donations would be greatly appreciated! Donations over $2 are tax deductible.
Thank you!
Donation Information
Phone: (02) 9716 0014
Bank Details:
BSB: 062257
Account No: 1008 9393
Account name: LEAD GROUP INCORPORATED NSW LEAD EDUCATION AND ABATEMENT FUND
Bank: Commonwealth Bank Australia, Summer Hill, NSW, 2130
The Lead Group Inc is endorsed as an income Tax Exempt Charitable entity under subdivision 50-B of the Income Tax Assessment Act 1997.
The Lead Education and Abatement Fund (LEAF) is a public fund listed on the Register of Environmental Organisations under Item 6.1.1 of subsection 30-55(1) of the Income Tax Assessment Act 1997 and is endorsed as a Deductible Gift Recipient under subdivision 30-BA of the Income Tax Assessment Act 1997.
and Wendy Evans our Administrator has just found online (see below) the details - including the brand name of the women's multivitamin with 15.3 micrograms of lead in the daily dose - it is the Vitamin Shoppe Multivitamins Especially for Women.
Just letting you know in case you're taking it! Stop now!!
Elizabeth O'Brien
Vitamins May Hurt Your Health
Vitamins may contain dangerous contaminants or different doses from the stated amounts. Sanja Gjenero, stock.xchng
MSN is running a feature about ConsumerLab.com's investigation into the purity of multivitamins. The lab looked at 21 brands of multivitamins for sale in the U.S. and Canada and found only 10 of these brands met the labeled claims or otherwise met quality standards. That doesn't have to mean anything earth-shattering. It could have been the other brands were close to meeting standards or had minor problems. However, the quality issues were ones that could actually hurt your health.
The Vitamin Shoppe Multivitamins Especially for Women were found to be contaminated with lead. Now, let's put this in perspective. Several calcium supplements run the risk of lead contamination, because lead and calcium participate in many of the same chemical reactions and are difficult to separate. That trace amounts of lead would be present might be expected. However, ConsumerLab.com reported a daily dose of this mulitvitamin contained a whopping 15.3 micrograms of lead (more than ten times the amount permitted without a warning in California). To make matters worse, though you got some bonus lead for your bucks, you only got 54% of the stated levels of calcium.
Another vitamin posed a different risk. Hero Nutritionals Yummi Bears, a kid's multivitamin, contained 216% of the labeled amount of vitamin A in the retinol form [5,400 International Units (IU)], which is considerably higher than the upper limit set by the Institute of Medicine of 2,000 IU for kids ages 1 to 3 and 3,000 IU for kids ages 4 to 8. Vitamin A is one of the vitamins where more is not better. Instead, too much vitamin A can weaken bones and cause liver damage.
Are these quality control issues? Yes, but I would have been surprised if the lab had found the vitamins met their stated claims. Why? For two reasons. First, vitamins aren't regulated by the same standards as medicine. They are considered 'supplements' and not 'drugs'. Your best defense against this is to buy a product from nationally-recognized reputable source with an interest in protecting its good name. The other reason I wouldn't expect vitamins to contain exactly what is listed on the label is simple chemistry. Vitamins, by their very nature, are reactive. The quantities listed in a product will change over the course of its shelf life. Your main protection here is to not take vitamins past their expiration date.
Should you take a multivitamin? Ask yourself whether the potential benefit outweighs the risk. If you are taking a major name brand multivitamin, you are probably getting approximately what is listed. Even then, expect some variation within the product and some degree of heavy-metal contamination with products that include minerals. These vitamins generally are safe, but don't take them automatically assuming they will help you.
Elizabeth,
Thank you so much for your prompt response. In answer to your question(s),
I was
exposed to lead while working in an old house (I am a contractor) and removing,
sanding
and generally breathing and/or ingesting lead paint particles. Needless to say
(I hope), I
am much more careful about my work habits and paying attention to the warnings
that
were already there for me.
As far as my levels go, I regret to say that I have only the one test to go
by as I was
dropped by my medical insurance on a technicality right after I was diagnosed
with a
"high" lead level. Regardless, I will be getting a new blood test for lead so I
can gauge
my progress, and I will most likeley have some more questions for you then.
Thanks again for your help and support; this has been a scary experience for
me and
it's good to be in contact with people who share my concerns.
sincerely, Saul
--- In LeadWorkers@yahoogroups.com, <egroup@...> wrote:
>
> Dear Saul,
> the good news is that your blood lead level will fall as long as you have
> identified the source/s and stopped the exposure. Sperm are apparently only
> affected by the blood lead level for the 4 months that it takes to make a
> sperm. We recommend therefore that you do not try to conceive until your
> blood lead level has been below 10 micrograms per decilitre (ug/dL) for a
> full four months prior to conception. As to how long it takes to get the
> blood lead level down to 10 ug/dL (ie how long will it be before you can
> start counting the four months), this is entirely dependent on the peak
> blood lead level and how chronic or acute the exposure was. In other words,
> if your blood lead level had been 26 ug/dL or higher for a full year before
> your blood lead level was tested (chronic exposure), then it would take a
> lot longer (unless you have chelation treatment) to fall to below 10 ug/dL
> than if your blood lead level was 8 ug/dL one week and 26 ug/dL for the next
> week (acute exposure) when you got it tested.
> If the result of 26 ug/dL was your first blood lead result then the best
> indicator as to how long your blood lead level might have been elevated is
> the RATE at which it falls or the length of time it takes to go below 10
> ug/dL. If it goes below 10 ug/dL in a matter of weeks then you know it was
> an acute exposure but if it takes months, you can be fairly sure it was a
> chronic exposure (unless there has been a failure to identify and eliminate
> sources and you are in fact continuing to be exposed to lead). Does that
> make sense?
> The bad news is that, according to the most comprehensive synthesis of
> research into lead, the "Draft Toxicological Profile for Lead" [Ref: Author:
> U.S. DEPARTMENT OF HEALTH AND HUMAN SERVICES, Public Health Service, Agency
> for Toxic Substances and Disease Registry (ATSDR), September 2005
> http://www.atsdr.cdc.gov/toxprofiles/tp13.pdf ]
> page 28 - "High-level exposure in men can damage the organs responsible for
> sperm production."
> Do you have other blood lead results to give the picture as to the severity
> of your exposure? Were you exposed to lead at work? If so, it would be
> interesting to know what the work was.
> Best regards
> Yours Sincerely
> Elizabeth O'Brien, Manager, Global Lead Advice & Support Service (GLASS) run
> by The LEAD Group Inc.
> PO Box 161 Summer Hill NSW 2130 Australia
> www.lead.org.au
>
>
> ----- Original Message -----
> From: "q22skidoo" <q22skidoo@...>
> To: <LeadWorkers@yahoogroups.com>
> Sent: Tuesday, September 12, 2006 3:01 PM
> Subject: [LeadWorkers] a question about plumbism and reproduction
>
>
> Hi. I'm a 35 year old male. Last year I was diagnosed with lead poisoning
> (my blood
> lead level was found to be >26 at the time I was tested). The symptoms come
> and go,
> but I'm dealing with them one day at a time.
>
> I have no children, but lately (for obvious reasons) I've been
> wondering about my
> ability to ever have them. Specifically, I'm wondering A. if a high blood
> lead level is a
> "death sentence" for a man's reproductive ability, and B. whether or not
> there are
> likely to be birth defects, assuming fertilization is still possible.
>
> I'd like to hear from anyone who has insight into this matter. Thanks.
>
>
>
>
>
>
> Yahoo! Groups Links
>
Dear Saul,
the good news is that your blood lead level will fall as long as you have
identified the source/s and stopped the exposure. Sperm are apparently only
affected by the blood lead level for the 4 months that it takes to make a
sperm. We recommend therefore that you do not try to conceive until your
blood lead level has been below 10 micrograms per decilitre (ug/dL) for a
full four months prior to conception. As to how long it takes to get the
blood lead level down to 10 ug/dL (ie how long will it be before you can
start counting the four months), this is entirely dependent on the peak
blood lead level and how chronic or acute the exposure was. In other words,
if your blood lead level had been 26 ug/dL or higher for a full year before
your blood lead level was tested (chronic exposure), then it would take a
lot longer (unless you have chelation treatment) to fall to below 10 ug/dL
than if your blood lead level was 8 ug/dL one week and 26 ug/dL for the next
week (acute exposure) when you got it tested.
If the result of 26 ug/dL was your first blood lead result then the best
indicator as to how long your blood lead level might have been elevated is
the RATE at which it falls or the length of time it takes to go below 10
ug/dL. If it goes below 10 ug/dL in a matter of weeks then you know it was
an acute exposure but if it takes months, you can be fairly sure it was a
chronic exposure (unless there has been a failure to identify and eliminate
sources and you are in fact continuing to be exposed to lead). Does that
make sense?
The bad news is that, according to the most comprehensive synthesis of
research into lead, the "Draft Toxicological Profile for Lead" [Ref: Author:
U.S. DEPARTMENT OF HEALTH AND HUMAN SERVICES, Public Health Service, Agency
for Toxic Substances and Disease Registry (ATSDR), September 2005
http://www.atsdr.cdc.gov/toxprofiles/tp13.pdf ]
page 28 - "High-level exposure in men can damage the organs responsible for
sperm production."
Do you have other blood lead results to give the picture as to the severity
of your exposure? Were you exposed to lead at work? If so, it would be
interesting to know what the work was.
Best regards
Yours Sincerely
Elizabeth O'Brien, Manager, Global Lead Advice & Support Service (GLASS) run
by The LEAD Group Inc.
PO Box 161 Summer Hill NSW 2130 Australia
www.lead.org.au
----- Original Message -----
From: "q22skidoo" <q22skidoo@...>
To: <LeadWorkers@yahoogroups.com>
Sent: Tuesday, September 12, 2006 3:01 PM
Subject: [LeadWorkers] a question about plumbism and reproduction
Hi. I'm a 35 year old male. Last year I was diagnosed with lead poisoning
(my blood
lead level was found to be >26 at the time I was tested). The symptoms come
and go,
but I'm dealing with them one day at a time.
I have no children, but lately (for obvious reasons) I've been
wondering about my
ability to ever have them. Specifically, I'm wondering A. if a high blood
lead level is a
"death sentence" for a man's reproductive ability, and B. whether or not
there are
likely to be birth defects, assuming fertilization is still possible.
I'd like to hear from anyone who has insight into this matter. Thanks.
Yahoo! Groups Links
Hi. I'm a 35 year old male. Last year I was diagnosed with lead poisoning (my
blood
lead level was found to be >26 at the time I was tested). The symptoms come and
go,
but I'm dealing with them one day at a time.
I have no children, but lately (for obvious reasons) I've been wondering
about my
ability to ever have them. Specifically, I'm wondering A. if a high blood lead
level is a
"death sentence" for a man's reproductive ability, and B. whether or not there
are
likely to be birth defects, assuming fertilization is still possible.
I'd like to hear from anyone who has insight into this matter. Thanks.
The following was kindly forwarded to me by a Leadnetter. Thanks.
Your really know the blood lead levels are high when they talk about diagnosing by symptoms: "Lead poisoning can be diagnosed from a blue line around the gums and in severe cases can cause convulsions, coma and death."
I'd still like to know the name of the company and whether it's Chinese or not.
Elizabeth O'Brien
From an Associated Press article online at MSNBC.com 9/6/06
HUNDREDS SUFFER LEAD POISONING IN CHINA Latest pollution disaster caused by a smelter in poverty-stricken Gansu
BEIJING - Hundreds of people in northwestern China have been hospitalized with lead poisoning that was likely caused by pollution from a nearby smelter, state media and local officials said Wednesday.
The poisonings in two villages in poverty-stricken Gansu province added to a string of recent pollution disasters in China that have prompted violent protests in some areas.
The first sign of trouble in the villages of Xinsi and Moba came on Aug. 18, when medical tests showed 10 people had high levels of lead in their blood, the Beijing Daily Messenger reported.
Health officials conducted checkups and "discovered that almost every family in the villages had the same kind of problem," or at least 879 residents, the newspaper said. The youngest victim was 5 months old.
"Children started feeling ill and their parents brought them to a local hospital," an official from Hui county, where the two villages are located, said by phone. "We suspect that they were sickened by pollution caused by a lead smelter nearby that discharged waste into the air."
The smelter was shut down and an investigation was under way, the official said. He refused to give his name or the name of the smelting company.
does anyone have any more detail on this story below? Like who owns the factory and what does it make? Electronics? Plastics? Fireworks? Pigments? Crystal? Leaded Glass? Computer recycling? So many options... Was it workers? Adults? Children?
Herald Sun Page 34 Thursday September 7 2006
Hundreds poisoned
BEIJING - At least 879 people in two Chinese villages have been admitted to hospital with lead poisoning, probably caused by airborne waste from a nearby lead factory, local officials said.
The poisonings in the poor, northwestern province of Gansu added to a string of recent pollution disasters that have prompted violent protests.